The Coexistence of Psoriasis and Lupus Erythematosus

Millns, John L.; Muller, Sigfrid A.

doi:10.1001/archderm.1980.01640300046015

Cited by 64 publications

(17 citation statements)

References 15 publications

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“…APAD is distinctive because of its distribution in the flexures. Generalized pustular and palmoplantar psoriasis has been described in association with systemic lupus erythematosus, 24–27 but pustular psoriasis is easily distinguishable from APAD and no APAD patients have had a personal or family history of psoriasis. Even though the pathophysiological relationship between APAD and autoimmunity remains to be elucidated, this new entity must be distinguished from other types of amicrobial pustulosis.…”

Section: Discussionmentioning

confidence: 99%

Amicrobial pustulosis associated with autoimmune diseases: healing with zinc supplementation

Bénéton¹,

Wolkenstein²,

Bagot³

et al. 2000

British Journal of Dermatology

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Section: Discussionmentioning

confidence: 99%

Amicrobial pustulosis associated with autoimmune diseases: healing with zinc supplementation

Bénéton¹,

Wolkenstein²,

Bagot³

et al. 2000

British Journal of Dermatology

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“…We would like to thank Prof. Manzke for his interest in our article [2]. His explanation about NNF is that in patients with AA it is rather a sign of epidermal atrophia visualizing the ectasia ofsubpapillary vessels in the nape than a concomitant skin abnormality.…”

Section: In Reply Nuchal Nevus Flammeusmentioning

confidence: 99%

Coexistence of Psoriasis vulgaris and Pemphigus foliaceus

Yokoo¹,

Oka²,

Ueki³

1989

Dermatology

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“…However, some patients with psoriasis experience aggravation of their disease by UV radiation or suffer from associated disorders that are known to be photosensitive [e.g. porphyria, chronic actinic dermatitis, systemic lupus erythematosus or polymorphic light eruption (PLE)] . Psoriasis induced by the so‐called Koebner response (or isomorphic phenomenon of Koebner) has been known for a long time .…”

Section: Introductionmentioning

confidence: 99%

Desired response to phototherapy vs photoaggravation in psoriasis: what makes the difference?

Wolf

Weger

Patra

et al. 2016

Experimental Dermatology

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Psoriasis commonly responds beneficially to UV radiation from natural sunlight or artificial sources. Therapeutic mechanisms include the proapoptotic and immunomodulating effects of UV, affecting many cells and involving a variety of pro-and anti-inflammatory cytokines, downregulating the Th17/IL-23 response with simultaneous induction of regulatory immune cells. However, exposure to UV radiation in a subset of psoriasis patients leads to exacerbation of the disease. We herein shed light on the predisposing factors of photosensitive psoriasis, including genetics (such as HLACw*0602 or CARD14), gender and coexisting photodermatoses such as polymorphic light eruption (PLE) in the context of potential molecular mechanisms behind therapeutic photoresponsiveness or photoaggravation. UV-induced damage/pathogenassociated molecular patterns, damage to self-coding RNA (signalling through Toll-like receptors), certain antimicrobial peptides and/or inflammasome activation may induce innate immunity, leading to psoriasis at the site of UV exposure when there is concomitant, predisposing resistance against UV-induced suppression of the adaptive immune response (like in PLE) that otherwise would act to reduce psoriasis. K E Y W O R D S

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The Coexistence of Psoriasis and Lupus Erythematosus

Cited by 64 publications

References 15 publications

Amicrobial pustulosis associated with autoimmune diseases: healing with zinc supplementation

Amicrobial pustulosis associated with autoimmune diseases: healing with zinc supplementation

Coexistence of Psoriasis vulgaris and Pemphigus foliaceus

Desired response to phototherapy vs photoaggravation in psoriasis: what makes the difference?

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