The effect of an oral contraceptive on serum lipids

Osman, Mohamed; Toppozada, Hussein K.; Ghanem, M. H.; Guergis, Fayez K.

doi:10.1016/0010-7824(72)90022-4

Cited by 16 publications

(3 citation statements)

References 25 publications

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“…correlating with duration of therapy and persisting after therapy stopped (37). 2) Increased levels of free fatty acis (38).…”

Section: General Category Description Of the Effect Possible Clinicalmentioning

confidence: 99%

“…Suppression of hypophyseal gonadotrophic Abolition of natural protection against activity (38). atherogenesis.…”

Section: Hormonal Balancementioning

confidence: 99%

“…atherogenesis. Lipid patterns in blood like after the menopause (38). Coagulation A) promotion of coagulability Increased clotting, leading to arterial 1) Thrombin mimetic effect (9,19) thromboemboli and venous thrombosis.…”

Section: Hormonal Balancementioning

confidence: 99%

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Changes in Blood Clotting Systems During Migraine Attacks

Kalendovsky

Austin²

1977

Headache

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SYNOPSIS The activity of various blood clotting systems was measured during attacks of migraine complicated by prolonged focal cerebral dysfunction and contrasted with those observed when the same and other patients had uncomplicated migraine. Results differed in the two groups. In patients with complicated migraine, plasma coagulability rose during both complicated and uncomplicated attacks. During a complicated migraine attack, platelet aggregability to ADP also rose in all but one patient. Platelet aggregability fell in the same patients when they had uncomplicated migraine. Plasma coagulability consistently increased more when the headache was associated with focal cerebral dysfunction. In patients with uncomplicated migraine, plasma coagulability dropped during the later phase of their attacks. This finding may reflect the release of endogenous heparin from mast cells and basophilic leucocytes. Platelet aggregability to ADP decreased in some but rose in others. The results support the hypothesis that prolonged focal cerebral dysfunction may reflect thromboembolic phenomena. The influence of estrogens and genetic factors on clotting systems is reviewed.

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“…correlating with duration of therapy and persisting after therapy stopped (37). 2) Increased levels of free fatty acis (38).…”

Section: General Category Description Of the Effect Possible Clinicalmentioning

confidence: 99%

“…Suppression of hypophyseal gonadotrophic Abolition of natural protection against activity (38). atherogenesis.…”

Section: Hormonal Balancementioning

confidence: 99%