The effect of oral tizanidine on postoperative pain relief after elective laparoscopic cholecystectomy

Abbasi, Saeed; Maghami, Elham; Zavareh, Sayyed Morteza Heidari Tabaei

doi:10.4103/2277-9175.175905

Cited by 25 publications

(46 citation statements)

References 6 publications

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“…Furthermore, the mean of heart rate and diastolic blood pressure in patients treated with tizanidine was lower than placebo group (11). Talakoub et al remarked that administration of tizanidine before operation in patients scheduled for elective laparoscopic cholecystectomy under general anesthesia improves postoperative analgesia, provides less pain intensity and decreases the amount of analgesic consumption (12). Henney et al carried out their study about pediatric safety of tizanidine.…”

Section: Discussionmentioning

confidence: 99%

Effect of oral tizanidine on prolongation of intrathecal lidocaine

Taheri

Hashemian

Noroozi

et al. 2018

J Emerg Pract Trauma

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Dear EditorScuba diving is associated with an important risk of developing decompression sickness secondary to formation of gas bubbles inside the body. The latter is formed mainly by nitrogen in the body on the diver's way to the surface (1,2). In some cases, it might injure the central nervous system. Several decompression cases that have been associated with neurologic symptoms are described in the literature; however, brain multi-infarct with lethal outcome has never been described. A 41-year-old male, came to the ER with clinical suspicion of decompression sickness (he dived for 15 minutes at a depth of 50 m). Among his health history, he was a heavy smoker, 30 minutes after leaving the water; he suddenly had dysarthria and vertigo. Upon admission, the patient had a poor general condition, Glasgow Coma Scale (GCS) 9 points, and horizontal nystagmus with right hemiparesis 4/5. Brain magnetic resonance imaging (MRI) demonstrated multiple cerebral and cerebellar infarctions. Echocardiography did not reveal the presence of patent foramen ovale (Figure 1). The patient was required to be transferred to intensive care unit and to a hyperbaric chamber session. He progressed to hemodynamic instability, dying within 36 hours after admission. Brain multi-infarct and decompression sickness© 2018 The Author(s). Published by Kerman University of Medical Sciences. This is an open-access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. Decompression sickness is thought to be associated with thrombotic events due to intravascular gas bubbles. It is accepted that mechanical abrasion to vessel's walls may induce endothelial dysfunction and activation of the blood cells that are responsible to start the inflammatory reaction, vasomotor dysfunction, platelet aggregation pathway and coagulopathy. This disease has been classified into two types: type I and II, based on the severity of signs and symptoms. Type I is a mild form that requires less recompression and treatment, type II is associated with neurologic symptoms (3). The latter requires an extensive therapy and may lead to significant neurologic sequelae. Our case report represents a type II decompression sickness. It is known that the delay to start recompression treatment represents a poor prognosis. Early start of hyperbaric therapy will reduce the size of the gas bubbles, improve perfusion and re-oxygenate ischemic tissue before late inflammatory processes might occur. These include cytokines, platelet activation and complement activation (4,5). Ethical issuesNot applicable. Authors' contributionsAll authors contributed equally. Jept Journal of Emergency Practice and TraumaLetter to Editor

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Section: Discussionmentioning

confidence: 99%

Effect of oral tizanidine on prolongation of intrathecal lidocaine

Taheri

Hashemian

Noroozi

et al. 2018

J Emerg Pract Trauma

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“…72 However, data are mounting for the perioperative benefit of tizanidine, including improvement in postoperative pain control, decreased dose requirements of midazolam, decreased opioid consumption, and/or hemodynamic stability. [73][74][75][76] Tizanidine clearance is significantly reduced when given concomitantly with ciprofloxacin and fluvoxamine and hence is contraindicated. Significant hypotension, bradycardia, and sedation can occur when given with other CYP1A2 inhibitors, such as oral contraceptives, acyclovir, amiodarone, verapamil, mexiletine, propafenone, cimetidine, and famotidine, among others.…”

Section: Tizanidinementioning

confidence: 99%

Preoperative Management of Opioid and Nonopioid Analgesics: Society for Perioperative Assessment and Quality Improvement (SPAQI) Consensus Statement

O'Rourke

Keshock

Boxhorn

et al. 2021

Mayo Clinic Proceedings

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“…It has been showed decreased pain intensity and decreased need for opioid analgesics. 47 Gabapentinoid: Gabapentinoids are analogs of γ-aminobutyric acid. They can bind to the α-2δ subunit of presynaptic calcium channels and therefore modulate glutamate expression.…”

Section: Pre-emptive Analgesiamentioning

confidence: 99%