2018
DOI: 10.13045/jar.2018.00024
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The Effects of Pulsatilla Koreana NAKAI Pharmacopuncture Therapy on the Chunchu (ST25) for Dextran Sulfate Sodium-Induced Colitis in Mice

Abstract: Background: This research was performed to investigate the effects of Pulsatilla Koreana NAKAI pharmacopuncture (PPA) therapy on intestinal disease in rats with dextran sulfate sodium (DSS)-induced colitis. Methods: The subjects were divided into five groups : A control group, saline group, pharmacopuncture group PPA1 (0.2 ㎎/ 1 kg/ 40 ㎕), pharmacopuncture group PPA2 (0.5 ㎎/ 1 kg/ 40 ㎕), and pharmacopuncture group PPA 3(1 ㎎/ 1 kg/ 40 ㎕). The experimental model of colitis was induced by infection of dextran sulf… Show more

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Cited by 3 publications
(2 citation statements)
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“…The level of IL‐6 is increased by the activation of the STAT3 signaling pathway, promoting the differentiation of CD4+ T cells into Th17 cells (Nagashima et al., 2018). TNF‐α increased intestinal mucosal damage through the caspase‐dependent pathway and the activation of the NF‐κB pathway, along with increased levels of IL‐1β, which acted on epithelial cells to trigger inflammation and hematopoiesis (Yang et al., 2018). In addition, IFN‐γ, an important proinflammatory cytokine, induces the production of other proinflammatory cytokines by promoting the differentiation of Th1 cells (Yang et al., 2018).…”
Section: Discussionmentioning
confidence: 99%
See 1 more Smart Citation
“…The level of IL‐6 is increased by the activation of the STAT3 signaling pathway, promoting the differentiation of CD4+ T cells into Th17 cells (Nagashima et al., 2018). TNF‐α increased intestinal mucosal damage through the caspase‐dependent pathway and the activation of the NF‐κB pathway, along with increased levels of IL‐1β, which acted on epithelial cells to trigger inflammation and hematopoiesis (Yang et al., 2018). In addition, IFN‐γ, an important proinflammatory cytokine, induces the production of other proinflammatory cytokines by promoting the differentiation of Th1 cells (Yang et al., 2018).…”
Section: Discussionmentioning
confidence: 99%
“…TNF‐α increased intestinal mucosal damage through the caspase‐dependent pathway and the activation of the NF‐κB pathway, along with increased levels of IL‐1β, which acted on epithelial cells to trigger inflammation and hematopoiesis (Yang et al., 2018). In addition, IFN‐γ, an important proinflammatory cytokine, induces the production of other proinflammatory cytokines by promoting the differentiation of Th1 cells (Yang et al., 2018). IL‐10 is an important anti‐inflammatory cytokine, and its level accurately reflects the host's immunity to inflammation.…”
Section: Discussionmentioning
confidence: 99%