The experimental production of parotid gland atrophy

Wallenborn, White McK.; Hsu, Ying-Che; Olinger, B.R.

doi:10.1288/00005537-196808000-00005

Cited by 13 publications

(4 citation statements)

References 16 publications

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“…In fact, many previously accepted methods of treatment such as radiotherapy and parasympathetic denervation are no longer commonly employed. 4,6,18,27,[29][30][31] Van Sickels 15 found that breaking down the parotid system into regions may facilitate decision making and predict postoperative problems with regard to traumatic parotid injuries. The 3 regions include Region A, or the glandular portion posterior to the masseter; Region B, or the duct and any glandular portion overlying the masseter; and Region C, which comprises the duct distal to the masseter and its associated papilla.…”

Section: Traumatic Injurymentioning

confidence: 99%

An Unusual Complication Following Rhytidectomy: Iatrogenic Parotid Injury Resulting in Parotid Fistula/Sialocele

Lawson

Kreyerman

Nahai

2012

Aesthetic Surgery Journal

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Rhytidectomy is a common surgical procedure performed by today's aesthetic surgeons. Newer trends and techniques leading to deeper and more aggressive dissection have placed the parotid gland at increased risk for injury during this procedure. Despite there being a relative abundance of literature on traumatic parotid injury, there is a relative paucity of information on iatrogenic parotid injury after rhytidectomy. In addition, there is no consensus on management of these complications. In this article, the authors discuss 3 case reports, review the relevant literature, and propose a treatment algorithm. Early diagnosis and appropriate treatment are essential to properly manage this complication.

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Section: Traumatic Injurymentioning

confidence: 99%

An Unusual Complication Following Rhytidectomy: Iatrogenic Parotid Injury Resulting in Parotid Fistula/Sialocele

Lawson

Kreyerman

Nahai

2012

Aesthetic Surgery Journal

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“…This modality, however, has been largely abandoned due to the adverse effects and potential for carcinogenesis associated with radiotherapy. [22][23][24][25] The major parasympathetic secretomotor fibers innervating the parotid gland are cholinergic, and use of anticholinergic medications, such as propantheline, exert a systemic effect that reduces secretions during the healing period. Unfortunately, the accompanying sideeffects of xerostomia, urinary retention, photophobia, tachycardia, and fatigue can make compliance difficult and limit their utility.…”

Section: Late Complicationsmentioning

confidence: 99%

Parotid Gland Trauma

et al. 2010

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Parotid trauma can lead to both short and long-term complications such as bleeding, infection, facial nerve injury, sialocele, and salivary fistula, resulting in pain and disfigurement. Facial injuries inferior to a line extended from the tragus to the upper lip should raise concern for parotid injury. These injuries can be stratified into three regions as they relate to the masseter muscle. Injuries posing the greatest risk of damage to Stensen's duct include those anterior to the posterior border of the masseter and necessitate exploration. When the duct is disrupted, emphasis should be placed on primary repair or re-creation of the papilla; however, proximal ductal lacerations can be treated by ligation of the proximal segment. Isolated parenchymal injury can be treated with more conservative means. Sialocele and salivary fistula can frequently be managed nonoperatively with antibiotics, pressure dressings, and serial aspiration. Anticholinergic medications and the injection of botulinum toxin represent additional measures before resorting to surgical therapies such as tympanic neurectomy or parotidectomy.

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“…Golding-Wood (1962) also ascribed a parotid secretory function to the chorda tympani and advocated resection of both tympanic fibres and the chorda tympani for the treatment of recurrent parotitis. Wallenborn and Olinger (1968) have demonstrated experimentally in rabbits that atrophy of the parotid gland can be produced by ligation of the parotid duct or by destruction of the tympanic plexus. Significant atrophy was noted to develop consistently about 25-29 weeks after surgical destruction of the tympanic plexus.…”

Section: Tympanic Neurectomymentioning

confidence: 99%