“…Several hypotheses also have been proposed for explaining why obese children are at risk of SDB, including reduction of the intrathoracic volume causing lower oxygen reserves [29], impaired ventilatory responses to hypoxia and hypercapnia, hypoventilation because of leptin resistance [30], and central apnea followed by narrowing or collapse of the upper airway [31]. In practice, an important concern is that obese children may have adenotonsillar hypertrophy [32,33], which may reduce the cross-sectional diameter of the upper airway [34]. However, the effect of adenotonsillar hypertrophy was not evaluated in our study.…”