The hemodynamic effects of induced supraventricular tachycardia in man.

Goldreyer, Bruce N.; Kastor, John A.; Kershbaum, Kenneth L.

doi:10.1161/01.cir.54.5.783

Cited by 90 publications

(29 citation statements)

References 15 publications

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“…Supraventricular tachycardia presenting as syncope without accompanying palpitations is probably rare [189] . Both non-invasive (transoesophageal) and invasive electrophysiological studies may be used to evaluate the haemodynamic effects of an induced tachycardia, especially if combined with administration of isoprenaline or atropine.…”

Section: Syncope In Patients With Bundle Branch Block (Impending Highmentioning

confidence: 99%

Guidelines on management (diagnosis and treatment) of syncope

Brignole¹,

Alboni²,

Benditt

et al. 2001

European Heart Journal

680

432

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Section: Syncope In Patients With Bundle Branch Block (Impending Highmentioning

confidence: 99%

Guidelines on management (diagnosis and treatment) of syncope

Brignole¹,

Alboni²,

Benditt

et al. 2001

European Heart Journal

680

432

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“…The increase of the heart rate produced by tachycardia was significantly greater in group A than in group B. The time for diastolic ventricular filling should be sortened to a greater extent by this striking tachycardia in group A, thereby leading to lower cardiac output and hence excessive hypotension [13]. The fall in blood pres sure in turn caused deterioration in renal blood flow, as indicated by the changes in p-aminohippurate clearance.…”

Section: Discussionmentioning

confidence: 90%

“…Since the atrial pressure rises during tachycardia [13], and the atrial natriuretic peptide is re leased by atrial distension [14], tachycardia-induced na triuresis might be caused by release of atrial natriuretic peptide. In order to clarify the role of this peptide in the natriuresis associated with tachycardia, several studies have been performed by inducing tachycardia [4][5][6][7], Al though many studies confirmed the rise in plasma levels of the peptide during tachycardia, the results pertaining to natriuresis were inconsistent.…”

Section: Discussionmentioning

confidence: 99%

Dominance of Blood Pressure in Natriuresis Associated with Supraventricular Tachycardia

Fujii

Kojima

Ohe

et al. 1991

Nephron

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Supraventricular tachycardia was induced in 10 patients by programmed cardiac stimulation through esophageal lead. Blood pressure, heart rate, renal function, and hormonal factors were measured before, during, and after tachycardia. The patients were divided into two groups, depending on whether antinatriuresis occurred during tachycardia; one group (n = 5) with antinatriuresis during tachycardia associated with a decrease in blood pressure and the other group (n = 5) with neither antinatriuresis nor changes in blood pressure. The urinary sodium excretion tended to increase after tachycardia only in the latter group. On the other hand, urine volume and free water clearance increased during or after tachycardia in both groups. Plasma levels of atrial natriuretic peptide significantly increased and the urinary vasopressin excretion significantly decreased during tachycardia in both groups. During tachycardia, natriuresis due to atrial natriuretic peptide secretion seems to be hampered by hypotension, but polyuria is preserved despite the fall in blood pressure probably related to suppression of vasopressin release.

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“…The patients were selected according to the following criteria: (1) history of documented symptomatic paroxysmal regular narrow QRS tachycardia; (2) normal PR interval (0.13-0.20 second) documented by 12-lead ECG; (3) electrophysiologic documentation of reciprocating supraventricular tachycardia due to reentry within the AV node or to reentry using a concealed accessory AV pathway;9-1" (4) absence of ventricular preexcitation (i.e., accessory AV, nodoventricular, or fasciculoventricular pathways with antegrade conduction properties) documented by detailed electrophysiologic study and by absence of a delta wave on the 12-lead ECG; (5) withdrawal of all cardioactive medications for at least three drug half-lives before intracardiac electrophysiologic study.…”

Section: Patient Selectionmentioning

confidence: 99%

Enhanced atrioventricular conduction in patients without preexcitation syndrome: relation to heart rate in paroxysmal reciprocating tachycardia.

et al. 1982

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We studied the electrophysiologic characteristics of atrioventricular (AV) nodal conduction in patients with reciprocating tachycardia (RT) without ventricular preexcitation, and the relation of these characteristics to RT cycle length (CL). Thirty-five symptomatic patients who had a normal PR interval (0.13-0.20 second) during sinus rhythm underwent detailed intracardiac electrophysiologic study during which ventricular preexcitation was excluded, and the RT mechanism was determined. RT was due to reentry using an accessory AV pathway capable of conduction only in the retrograde direction (concealed AP) in 13 patients (37%) and to reentry within the AV node in 22 (63%). Dynamic properties of AV conduction (assessed by degree of AH prolongation during progressive increase in atrial pacing rate) were normally distributed (p less then 0.005); 12 patients (34%) fulfilled the criteria for enhanced AV conduction (EAVC). The patients with EAVC had a shorter RT CL than did patients without EAVC (294 +/- 43.3 msec vs 360 +/- 68.1 msec, p less than 0.01). However, CL differences were primarily due to the influence of EAVC in the subgroup of patients with RT using a concealed AP (EAVC CL, 274 +/- 35.1 msec; without EAVC, 326 +/- 15.7 msec, p less than 0.005). The RT CL in patients with reentry within AV node was not measureable influenced by concomitant EAVC (EAVC, 314 +/- 43.8 msec; without EAVC, 376 +/- 76.8 msec) (NS). This study suggests that despite the presence of a normal PR interval during sinus rhythm, dynamic AV conduction responses can vary widely in patients with RT. In patients with RT using a concealed AP, but not in those with reentry within the AV node, coexisting diminished physiologic AV conduction slowing may be associated with more rapid tachycardia rates.

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The hemodynamic effects of induced supraventricular tachycardia in man.

Cited by 90 publications

References 15 publications

Guidelines on management (diagnosis and treatment) of syncope

Guidelines on management (diagnosis and treatment) of syncope

Dominance of Blood Pressure in Natriuresis Associated with Supraventricular Tachycardia

Enhanced atrioventricular conduction in patients without preexcitation syndrome: relation to heart rate in paroxysmal reciprocating tachycardia.

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