The Pharmacologic Intensification of the Water Dissociation Process, or Human Photosynthesis, and Its Effect over the Recovery Mechanisms in Tissues Affected by Bloodshed of Diverse Etiology

Herrera, Arturo Solís; Esparza, María del Carmen Arias; Esquivel, J. Jesús Alvarado; Miranda, Graciela Landín; Arias, Ruth Isabel Solís; Arias, Paola Eugenia Solís; Arias, Martha Patricia Solís

doi:10.4236/ijcm.2011.23058

Cited by 4 publications

(2 citation statements)

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“…The remarkable beneficial effects of enhancing human photosynthesis in different diseases have been reported in other publications [14]. Initially we thought that the body's ability to transform light energy into chemical energy only happened in the retina, but as we went deeper into the subject, we realized that each and every cell of the body works on the same principle: We are shaped by billions of units (cells) energetically independent.…”

Section: Discussionmentioning

confidence: 70%

Neuron Cell Degeneration Results from Perturbed Mitochondria or <i>Vice Versa</i>?

Solís-Herrera¹,

Arias-Esparza²,

Solís-Arias³

2015

WJNS

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Mitochondrial disease has important implications for numerous functions that affect different cells lines and tissues. The mitochondrion is an organelle that draws much attention from researchers, but their study is hampered by changing in number, size, shape, and location among other things. To date, it is not understood if mitochondrial alterations could be considered to be downstream effects or are the root causes of diseases classified as neurodegenerative. It is an unresolved enigma if mitochondrion form, size, location, and number become crucial to the correct functioning of distinct cells lines and tissues, besides the role of reactive oxygen species (ROS) generated during the metabolism of glucose. The unexpected role of melanin to transform radiative energy directly into chemical energy by dissociation of the water molecule marks a milestone in the study of mitochondrial diseases.

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Section: Discussionmentioning

confidence: 70%

Neuron Cell Degeneration Results from Perturbed Mitochondria or <i>Vice Versa</i>?

Solís-Herrera¹,

Arias-Esparza²,

Solís-Arias³

2015

WJNS

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“…a failure in light/melanin/water system would be a cause rather than effect of AD has been proposed (26) . The decrease in melanins ability to dissociate water (human photosynthesis) in age-related macular degeneration (AMD) (27) and or AD has been proposed to be a cause of these diseases is a simplistic overview of the bioenegetic mechanism related to these diseases.…”

Section: Discussionmentioning

confidence: 99%

Evaluation of Melatonin, and Adipokines in Patients with Alzheimer’s Disease

Sarhat¹

2015

European Journal of Medicine. Series B

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Alzheimer's disease (AD) is one of the most common causes for the development of Dementia in the elderly. In past two decades there has been abundant research in pathogenesis of AD and possible prevention and treatment. The objective of this study is to evaluate the level of melatonin, and adipokines in patients with AD and control group. The study included 52 patients with AD and 52 healthy subject as control group. The results showed that serum melatonin, leptin, adiponectin, levels of patients with AD were significantly lower than the levels found for control participants (P < 0.0001). Patients with AD had significantly higher mean serum ghrelin, and resistin levels than the controls (P < 0.0001), there was positive correlation between adiponectin with leptin, and ghrelin. In conclusion, the results of this study showed that Circulating melatonin, leptin, and adiponectin were associated with a reduced incidence of AD, Resistin levels may be considered as a predictor of AD and it may predict activation of the immune system in AD pathophysiology.

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The Beneficial Effects of QIAPI 1® against Pentavalent Arsenic-Induced Lung Toxicity: A Hypothetical Model for SARS CoV2-I nduced Lung Toxicity

Herrera

Beeraka

Sinelnikov

et al. 2022

CPB

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: Exposure to environmental toxicants such as Arsenic (As) can result in As-induced alterations in immune regulators. Consequently, people who are more prone to viral infections like influenza A or B, H1N1, SARS CoV (Severe Acute Respiratory Syndrome Coronavirus), and SARS CoV2 may develop susceptibility to immune responses in their lungs because our previous reports delineated the ability of QIAPI 1®, a melanin precursor, to dissociate water molecules with simultaneous therapeutic efficacy against central nervous system (CNS) diseases, retinopathy, and As-induced renal toxicity. Given the commonalities of lung pathology of SARS CoV and As-induced toxicity, the aim of this study is to decipher the efficacy of QIAPI 1® against pentavalent As-induced lung toxicity by examining the pulmonary pathology. Hematoxylin & Eosin (H&E) staining was used for ascertaining the lung pathology in Wistar rat models. Animals were divided into 3 groups: control group, group treated with pentavalent As, and a group treated with pentavalent As and QIAPI 1®. There were no significant changes in lung histopathology in the control group as indicated by intact morphology. As-treated group revealed damage to the histoarchitecture with pulmonary edema, interstitial fibrosis, diffuse alveolar damage, Bronchiolitis obliterans organizing pneumonia (BOOP)-lesions, formation of hyaline membrane, multinucleated giant pneumocytes, atypical pneumocytes, inflammatory cell infiltration, and interstitial edema. The group treated with As and QIAPI 1® significantly associated with mitigated histological signs of lung inflammation induced by Arsenic. Therefore, QIAPI 1® can be recommended as antagonistic to As-induced lung toxicity. In conclusion, this model could be preferred as a hypothetical model to examine the efficacy of QIAPI 1® in SARS CoV2-induced pulmonary damage. Future studies are warranted to delineate the efficacy of QIAPI 1® against SARS CoV and SARS CoV2 lung pathology.

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The Pharmacologic Intensification of the Water Dissociation Process, or Human Photosynthesis, and Its Effect over the Recovery Mechanisms in Tissues Affected by Bloodshed of Diverse Etiology

Cited by 4 publications

References 2 publications

Neuron Cell Degeneration Results from Perturbed Mitochondria or <i>Vice Versa</i>?

Neuron Cell Degeneration Results from Perturbed Mitochondria or <i>Vice Versa</i>?

Evaluation of Melatonin, and Adipokines in Patients with Alzheimer’s Disease

The Beneficial Effects of QIAPI 1® against Pentavalent Arsenic-Induced Lung Toxicity: A Hypothetical Model for SARS CoV2-I nduced Lung Toxicity

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The Pharmacologic Intensification of the Water Dissociation Process, or Human Photosynthesis, and Its Effect over the Recovery Mechanisms in Tissues Affected by Bloodshed of Diverse Etiology

Cited by 4 publications

References 2 publications

Neuron Cell Degeneration Results from Perturbed Mitochondria or &lt;i&gt;Vice Versa&lt;/i&gt;?

Neuron Cell Degeneration Results from Perturbed Mitochondria or &lt;i&gt;Vice Versa&lt;/i&gt;?

Evaluation of Melatonin, and Adipokines in Patients with Alzheimer’s Disease

The Beneficial Effects of QIAPI 1® against Pentavalent Arsenic-Induced Lung Toxicity: A Hypothetical Model for SARS CoV2-I nduced Lung Toxicity

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Product

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Neuron Cell Degeneration Results from Perturbed Mitochondria or <i>Vice Versa</i>?

Neuron Cell Degeneration Results from Perturbed Mitochondria or <i>Vice Versa</i>?