E 6 r s Bajusz
Bio-Research Institute, Cambridge, M a s s .Since the discovery by Deming and Luetscherl of increased sodiumretaining activity in urine from patients with congestive heart failure, the role of aldosterone in the pathogenesis of cardia edema has been extensively investigated. It is generally believed that, in an effort to maintain blood pressure, aldosterone promotes sodium reabsorption, leading to increased blood volume and eventually to edema.2 During the course of these investigations,