2013
DOI: 10.20538/1682-0363-2013-6-54-61
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The ROLE OF TUMOR NECROSIS FACTOR-α G–308A, INTERLEUKIN-1β C–511T AND INTERLEUKIN-10 G–1082A GENE POLYMORPHISMS IN THE DEVELOPMENT OF SLOWLYRESOLVED COURSE OF COMMUNITY-ACQUIRED PNEUMONIA

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