The Total Work of Breathing in Normal and Obese Men*

Sharp, John T.; Henry, J. P.; Sweany, Stanford K.; Meadows, William R.; Pietras, Raymond J.

doi:10.1172/jci104957

Cited by 299 publications

(183 citation statements)

References 25 publications

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“…7 The work of breathing is markedly increased in patients with severe obesity. 11 Maximum voluntary ventilation as well as maximal inspiratory pressure (PImax) can also be low, [6][7][8][9][10] reflecting a decreased inspiratory muscle performance.…”

Section: (Ohs)mentioning

confidence: 99%

Physiologic correlates of dyspnea in patients with morbid obesity

et al. 2006

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Objective: Mechanisms of dyspnea in obesity remain unclear. This study was undertaken to determine the relationships between dyspnea and pulmonary function including inspiratory muscle endurance (IME) in morbidly obese patients before bariatric surgery. Research methods and procedures: Fifty-five patients with a mean7s.d. body mass index (BMI) of 49.477.0 kg/m 2 were included. Dyspnea was evaluated by the Baseline Dyspnea Index (BDI; 0-12, 0 ¼ maximal dyspnea). Pulmonary function tests included a plethysmography, maximal inspiratory pressure (PImax) and IME was assessed by the incremental threshold loading test, determining the maximal pressure sustained for 2 min (Plim 2 ) and Plim 2 /PImax ratio. Patients were classified according to their BMI in two groups: BMI p49 (n ¼ 27) and 449 kg/m 2 (n ¼ 28

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Section: (Ohs)mentioning

confidence: 99%

Physiologic correlates of dyspnea in patients with morbid obesity

et al. 2006

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“…Clearly the literature has so far presented a great discrepancy in the subject, and the controversies over it remain. In conscious patients, the investigations using different methods report a decrease in thoracic compliance 4,27 . In contrast, Suratt et al 28 compared obese and non-obese conscious patients and did not find any correlation between BMI and thoracic wall compliance.…”

Section: Discussionmentioning

confidence: 99%

Estudo dos volumes pulmonares e da mobilidade toracoabdominal de portadoras de obesidade mórbida, submetidas à cirurgia bariátrica, tratadas com duas diferentes técnicas de fisioterapia

Costa

Forti

et al. 2009

Rev. bras. fisioter.

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© Revista Brasileira de FisioterapiaStudy on pulmonary volumes and thoracoabdominal mobility in morbidly obese women undergoing bariatric surgery, treated with two different physical therapy methods ), before surgery and 15 and 30 days after surgery. The candidates were evaluated with regard to measurements of inspiratory reserve volume (IRV), expiratory reserve volume (ERV), inspiratory capacity (IC) and thoracoabdominal mobility, by means of spirometry and cirtometry, respectively. CRP consisted of diaphragmatic respiratory exercises, deep fractionated inspiration and respiratory exercises associated with upper limb movement. One set of 10 repetitions of each exercise was carried out twice daily while hospitalized. For TEDS, two electrodes were placed on the parasternal region, next to the xiphoid process, and another two between the sixth and seventh intercostal spaces, bilaterally on the anterior axillary lines. Friedman's test was used to compare repeated measures within groups, and the Mann-Whitney test for comparisons between groups. P values <0.05 were taken to be statistically significant. Results: The IRV, ERV and thoracoabdominal mobility measurements increased significantly in the CRP+TEDS group. In contrast, the IC measurements decreased significantly both in the CRP and in the CRP+TEDS groups. Conclusion: The obese women who underwent bariatric surgery and received postoperative CRP+TEDS presented greater gains in some of the pulmonary volumes and improvements in the amplitude of respiratory movements.

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“…When compared to subjects who are of normal weight, respiratory system compliance has been shown to be about 20% less in eucapnic obese individuals and almost 60% less in patients with OHS [23,26]. Small airway closure during exhalation can occur while breathing at low lung volumes, thus creating expiratory flow limitation and intrinsic positive and expiratory pressure (PEEPi) [27][28][29].…”

Section: Respiratory Mechanicsmentioning

confidence: 99%

Obesity Hypoventilation Syndrome

2010

IJPM

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Obesity hypoventilation syndrome is a respiratory consequence of morbid obesity that is characterized by alveolar hypoventilation during sleep and wakefulness. The disorder involves a complex interaction between impaired respiratory mechanics, ventilatory drive and sleepdisordered breathing. Early diagnosis and treatment is important, because delay in treatment is associated with significant mortality and morbidity. Available treatment options include noninvasive positive airway pressure (PAP) therapies and weight loss. There is limited long-term data regarding the effectiveness of such therapies. This review outlines the current concepts of clinical presentation, diagnostic and management strategies to help identify and treat patients with obesityhypoventilation syndromes.

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The Total Work of Breathing in Normal and Obese Men*

Cited by 299 publications

References 25 publications

Physiologic correlates of dyspnea in patients with morbid obesity

Physiologic correlates of dyspnea in patients with morbid obesity

Estudo dos volumes pulmonares e da mobilidade toracoabdominal de portadoras de obesidade mórbida, submetidas à cirurgia bariátrica, tratadas com duas diferentes técnicas de fisioterapia

Obesity Hypoventilation Syndrome

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