2022
DOI: 10.1016/j.ijcha.2022.101026
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The tricuspid annulus in amyloidosis with cardiac involvement: Detailed analysis from the three-dimensional speckle tracking echocardiographic MAGYAR-Path Study

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Cited by 3 publications
(3 citation statements)
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“…Of note, more unexpected presentations of MR in CA, such as ruptured chordae, have also been published [ 35 ]. Moreover, CA is associated with distinct LV geometry changes, which in turn can also cause insufficient mitral valve closure [ 28 , 36 , 37 ]. Finally, left atrial enlargement represents a hallmark sign of restrictive cardiomyopathies, often leading to mitral valve annular dilatation and atrial functional MR [ 32 ].…”
Section: Mitral Regurgitationmentioning
confidence: 99%
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“…Of note, more unexpected presentations of MR in CA, such as ruptured chordae, have also been published [ 35 ]. Moreover, CA is associated with distinct LV geometry changes, which in turn can also cause insufficient mitral valve closure [ 28 , 36 , 37 ]. Finally, left atrial enlargement represents a hallmark sign of restrictive cardiomyopathies, often leading to mitral valve annular dilatation and atrial functional MR [ 32 ].…”
Section: Mitral Regurgitationmentioning
confidence: 99%
“…Furthermore, our literature review yielded only two case reports on concomitant AV valve stenosis in CA (one AL and one Serum amyloid A amyloidosis) [ 47 , 48 ]. This low prevalence may partly be explained by the AV annulus dilatation, which is commonly observed in CA and counteracts possible stenotic processes [ 37 ].…”
Section: Mitral and Tricuspid Stenosismentioning
confidence: 99%
“…(35) Moreover, CA is associated with distinct LV geometry changes which in turn can also cause insufficient mitral valve closure. (28,36,37) Finally, left atrial enlargement represents a hallmark sign of restrictive cardiomyopathies, often leading to mitral valve annular dilatation and atrial functional MR. (32) Mixed etiologies may also occur. Thus, MR in the context of CA is indeed a rather complex disease, which is underlined by the fact that it can present as Carpentier classes I-III.…”
Section: Pathophysiological Aspectsmentioning
confidence: 99%