Molar incisor hypomineralization (MIH) is a qualitative developmental defect of enamel affecting a minimum of one first permanent molar, often with involvement of the permanent incisors. 1 Despite MIH having been detected among ancient populations, 2,3 it was first identified in 1987 by Koch and defined in 2001. 1,4 MIH lesions differ considerably from sound enamel. Microscopically, these lesions present disorganized enamel prisms and large inter-prismatic spaces occupied by a protein-rich matrix associated with decreased mineral density, hardness, and fracture resistance. This explains the increased surface porosity and post-eruptive breakdown (PEB) of the enamel surface (especially in severe lesions) and the greater susceptibility to dental caries; and also why affected teeth are often hypersensitive. [5][6][7][8][9][10]