Third generation personal communication systems

We study the class of complex algebraic K3 surfaces admitting an embedding of H ⊕ E8 ⊕ E8 inside the Néron-Severi lattice. These special K3 surfaces are classified by a pair of modular invariants, in the same manner that elliptic curves over C are classified by the J-invariant. Via the canonical Shioda-Inose structure we construct a geometric correspondence relating K3 surfaces of the above type with abelian surfaces realized as cartesian products of two elliptic curves. We then use this correspondence to determine explicit formulas for the modular invariants.

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“…A very interesting alternative arithmetic approach to the above questions, by Elkies and Kumar, has been communicated to the authors [22].…”

Section: Introductionmentioning

confidence: 99%

Modular invariants for lattice polarized K3 surfaces

Clingher¹,

Doran²

2007

Michigan Math. J.

147

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“…Environmental factors include smoking and obesity , and both have been shown to be associated with dysbiotic subgingival and intestinal communities, respectively. In fact, it has recently been found that smoking creates a subgingival microbiome in healthy sites that more closely resembles that of diseased sites, suggesting that smoking creates ‘an at‐risk‐for‐harm environment’ to create periodontitis . Diabetes is also associated with periodontitis and it has been postulated that a more complete analysis of the periodontal microbiome is necessary to determine if this disease is associated with a dysbiotic periodontal community.…”

Section: What Causes Dysbiosis?mentioning

confidence: 99%

Microbial protection and virulence in periodontal tissue as a function of polymicrobial communities: symbiosis and dysbiosis

Roberts¹,

Darveau²

2015

Periodontology 2000

105

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This review discusses polymicrobial interactions with the host in both health and disease. As our ability improves to identify specific bacterial clonal types both with respect to abundance and location in the oral biofilm we will learn more concerning their contribution to both oral health and disease. Recent studies examining host-bacterial interactions have revealed that commensal bacteria not only protect the host simply by niche occupation, but that bacterial interactions with host tissue can promote the development of proper tissue structure and function. These data indicate that our host-associated polymicrobial communities, such as those found in the oral cavity, co-evolved with us and have become an integral part of who we are. Understanding the microbial community factors that underpin associations with host tissue that contribute to periodontal health may also reveal how dysbiotic periodontopathic oral communities disrupt normal periodontal tissue functions in disease. A disruption of the oral microbial community creates dysbiosis, either by overgrowth of specific or nonspecific microorganisms or changes in the local host response where the community can now support a disease state. Dysbiosis provides the link between systemic changes (e.g., diabetes), exogenous risk factors (e.g., smoking), and the dysbiotic community and can drive the periodontal tissue destruction. Many other risk factors associated with periodontal disease such as stress, aging, and genetics also likely affect the microbial community, and more research is needed utilizing sophisticated bacterial taxonomic techniques to better elucidate these effects on the microbiome and to develop strategies to target the dysbiotic mechanisms and improve periodontal health.

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“…The various proposed network-level approaches differ in the placement of the home address functionality and in the protocol adopted to update the home address information [7]. The two most diffused proposals are Mobile IP, that is backward compatible with IP, but cannot achieve optimal routing (triangle routing problem [7]), and IPv6, that provides both acceptable performance and excellent scalability, but whose process of acceptance seems still long, as for any new protocol [26].…”

Section: Related Workmentioning

confidence: 99%

“…In particular, terminal mobility permits mobile devices to roam in the global system while maintaining the same ability of service access and provision, and user mobility provides users a uniform vision of their preferred working environment independently of their current points of attachment [20,26].…”

Section: Introductionmentioning

confidence: 99%

A mobile agent infrastructure for the mobility support

Bellavista

Corradi

Stefanelli

2000

Proceedings of the 2000 ACM Symposium on Applied Computing - Volume 2

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The mobility of terminals and users is a crucial issue in the open global system represented by the Internet. Supporting terminal and user mobility requires a middleware infrastructure capable of efficiently answering the needs of scalable resource discovery, of security and interoperability, of Quality of Service (QoS) monitoring and adaptation. Some proposals address mobility at the network level and some others focus on application-level solutions. By following the latter approach, the paper proposes the adoption of the mobile agent technology to model and implement mobility. In particular, the paper concentrates on the components and modules implemented in the SOMA mobile agent programming framework to specifically support terminal and user mobility. The SOMA tracing and discovery system extends the SOMA basic naming service to identify and keep track of all mobile entities in the environment. The SOMA QoS adaptation support exploits the functionality of the SOMA monitoring tools and permits to dynamically adjust service provision in response to the changing network and nodes conditions. These features are integrated in a mobility add-on module that pursues also the goals of security and interoperability when moving users and roaming terminals.

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Third generation personal communication systems

Cited by 8 publications

References 0 publications

Modular invariants for lattice polarized K3 surfaces

Modular invariants for lattice polarized K3 surfaces

Microbial protection and virulence in periodontal tissue as a function of polymicrobial communities: symbiosis and dysbiosis

A mobile agent infrastructure for the mobility support

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