1978
DOI: 10.7326/0003-4819-89-5-647
|View full text |Cite
|
Sign up to set email alerts
|

Thrombotic Thrombocytopenic Purpura Fails to Respond to Fresh Frozen Plasma Infusion

Help me understand this report

Search citation statements

Order By: Relevance

Paper Sections

Select...
4
1

Citation Types

2
19
0

Year Published

1980
1980
2010
2010

Publication Types

Select...
10

Relationship

0
10

Authors

Journals

citations
Cited by 50 publications
(21 citation statements)
references
References 8 publications
2
19
0
Order By: Relevance
“…In these reports the increased PAIgG resolved as the patient recovered. In other studies the level of PAIgG has, however, been reported as normal (Ansell et al, 1978;Cines & Schreiber, 1979). Increased PA-C3 has also been reported (Garvey & Freedman, 1983).…”
Section: Discussionmentioning
confidence: 82%
“…In these reports the increased PAIgG resolved as the patient recovered. In other studies the level of PAIgG has, however, been reported as normal (Ansell et al, 1978;Cines & Schreiber, 1979). Increased PA-C3 has also been reported (Garvey & Freedman, 1983).…”
Section: Discussionmentioning
confidence: 82%
“…Later on during the same year, Bukowski et al [30] introduced plasma exchange as a first-line treatment, which has significantly improved the outcome of this rare yet potentially fatal hematological disease. Several studies have shown the superiority of plasma exchange over FFP infusion in treating TTP [3,31,32]. The American Association of Blood Banks (AABB) and the American Society for Apheresis (ASFA) classifies therapeutic plasma exchange as indication category I in the treatment of TTP.…”
Section: Discussionmentioning
confidence: 99%
“…A necropsy study of 50 patients with neurological manifestations of SLE showed pathological central nervous system alterations in 50% of patients. In the final stages of their disease, 14 patients presented a clinical syndrome of thrombotic thrombocytopenic purpura; in half of these pathological thrombotic thrombocytopenic purpura changes in the central nervous system (occlusion of arterioles and capillaries with platelet-fibrin thrombi, segmental subendothelial hyalin deposits, and arteriolar microaneurysms) were observed. In ten of the remaining patients, cerebral infarctions secondary to embolism of cardiac origin (Libman-Sacks endocarditis in five, chronic valvulitis in two, heart mural thrombus in two), were reported.…”
Section: Discussionmentioning
confidence: 99%