Transient Increase in Wall Thickness of the Left Ventricular Apex after Stunned Myocardium: A Case Report.

Iga, Kanji; Kitaguchi, Katsuji; Hori, Kenjiro; Matumura, Tadashi; Gen, Hiromitu; Tomonaga, Go; Tamamura, Toshitake

doi:10.2169/internalmedicine.31.122

Cited by 3 publications

(3 citation statements)

References 3 publications

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“…4 This hypertrophy could be interpreted as to be due to transitory interstitial edema. [1][2][3][4]6 In our case, a left ventricular hypertrophy wall was detected on the sixth day of the illness Figure 3 was performed in the cardiology laboratory, not in the ICU, using different equipment but the same technique.) and disappeared in the next few days.…”

Section: Discussionmentioning

confidence: 69%

Acute Myocarditis and Left Ventricular “Hypertrophy”

et al. 2000

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Echocardiographic abnormalities of myocarditis are polymorphous and nonspecific. The presence of increased ventricular wall thickness, typically transient, is an infrequent finding in myocarditis that can correspond to an improvement in the clinical status of the patient and the ejection fraction. We report the case of a patient with acute myocarditis and the echocardiographic abnormalities observed during the course of his myocarditis: transient left ventricular "hypertrophy" associated with improvement of the left ventricular function.

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Section: Discussionmentioning

confidence: 69%

Acute Myocarditis and Left Ventricular “Hypertrophy”

et al. 2000

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“…In contrast, apical wall thickness gradually increased, and apical hypertrophy appeared over a few weeks to months. Subsequently, the apical wall thickness gradually decreased and returned to normal thickness over a few months to a year [1][2][3][4]. In this case, the left ventricular wall motion abnormalities improved on day 3, and apical hypertrophy appeared on day 20, and returned to normal on day 51.…”

Section: Discussionmentioning

confidence: 72%

“…A case of transient apical hypertrophy associated with coronary vasospasm is rare. Few clinical cases have reported transient apical hypertrophy following takotsubo cardiomyopathy and acute myocarditis in addition to coronary vasospasm [1][2][3][4].…”

Section: Discussionmentioning

confidence: 99%

A case of transient apical hypertrophy associated with coronary vasospasm

Awaya

Moroi

Takagi

et al. 2017

Journal of Cardiology Cases

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Transient increase in wall thickness of the left ventricular apex during recovery from “ampulla” cardiomyopathy

Tanaka¹,

Harada²,

Shiraishi³

et al. 2003

Ann Nucl Med

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"Ampulla" cardiomyopathy is a syndrome characterized by transient abnormal left ventricular wall motion with hypokinesia around the apical area and hyperkinesia at the basal area, without any detectable coronary lesion. Two cases of transient wall thickening of the left ventricular apex during recovery from "ampulla" cardiomyopathy are described. Apical wall thickening was documented by left ventriculography, echocardiography, and thallium (201Tl) single-photon emission computed tomography (SPECT) during the recovery phase. The thickness of the apical wall subsequently returned to normal. Both patients underwent provocation tests. Coronary spasms were positive. This transient increase in left ventricular apical volume may have been caused by myocardial inflammation secondary to "ampulla" cardiomyopathy.

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Transient Increase in Wall Thickness of the Left Ventricular Apex after Stunned Myocardium: A Case Report.

Cited by 3 publications

References 3 publications

Acute Myocarditis and Left Ventricular “Hypertrophy”

Acute Myocarditis and Left Ventricular “Hypertrophy”

A case of transient apical hypertrophy associated with coronary vasospasm

Transient increase in wall thickness of the left ventricular apex during recovery from “ampulla” cardiomyopathy

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