2013
DOI: 10.1111/echo.12139
|View full text |Cite
|
Sign up to set email alerts
|

Two Cases of Reversible Left Ventricular Hypertrophy during Recovery from Takotsubo Cardiomyopathy

Abstract: We report 2 cases of reversible ventricular hypertrophy in patients with takotsubo cardiomyopathy (stress-induced cardiomyopathy) during recovery of cardiac function. The first case involved a 72-year-old woman who presented with cerebral infarction. On admission, an elevated troponin I and decreased apical wall motion were observed with normal myocardial perfusion imaging. The second case involved a 79-year-old woman who presented with angina, anxiety resulting from emotional stress, slightly decreased apical… Show more

Help me understand this report

Search citation statements

Order By: Relevance

Paper Sections

Select...
2
1
1

Citation Types

0
16
0

Year Published

2013
2013
2024
2024

Publication Types

Select...
8

Relationship

1
7

Authors

Journals

citations
Cited by 17 publications
(16 citation statements)
references
References 7 publications
0
16
0
Order By: Relevance
“…We have observed this finding in several patients with SCM during recovery. There have also been several case reports regarding this phenomenon [9][10][11][12]; however, the incidence and clinical significance of this finding had not been studied. The results Data are expressed as number (%) or mean ± standard deviation.…”
Section: Discussionmentioning
confidence: 99%
See 1 more Smart Citation
“…We have observed this finding in several patients with SCM during recovery. There have also been several case reports regarding this phenomenon [9][10][11][12]; however, the incidence and clinical significance of this finding had not been studied. The results Data are expressed as number (%) or mean ± standard deviation.…”
Section: Discussionmentioning
confidence: 99%
“…Although reports on SCM are increasing, the nature of SCM remains ill-defined. Recently, transient apical wall thickening (TAWT), which mimics apical hypertrophic cardiomyopathy during recovery from SCM, has been reported [9][10][11][12]. This phenomenon developed during recovery of SCM patients who showed a marked increase in LV apical wall thickness that subsequently resolved [9][10][11][12].…”
Section: Introductionmentioning
confidence: 99%
“…In humans, catecholamines may cause toxic myocarditis in cases of phaeocromocytoma or stress‐induced (Takotsubo) cardiomyopathy due to an exaggerated sympathetic stimulation . Takotsubo cardiomyopathy is a transient left ventricular dysfunction most frequently characterized by an apical “ballooning” phenotype in people, but myocardial edema mimicking HCM has also been described in Takotsubo cases, and differentiation from true HCM requires advanced cross‐sectional imaging . TMT could then potentially be caused by an emotional or physical stress associated with the reported antecedent events.…”
Section: Discussionmentioning
confidence: 99%
“…4 Kato and colleagues 3 observed apical hypertrophy of the LV at approximately 3 weeks after onset, when the wall motion had improved; the ventricular wall gradually became thinner, and the transient apical hypertrophy was attributed to hypertrophic signaling in the myocardium, which was stimulated by catecholamines. 3 Myocardial edema with a hypertrophic LV apex has been reproducibly detected on cardiac magnetic resonance images 4 and echocardiograms. Whether the LV hypertrophy 1 represents apical HCM or takotsubo-induced myocardial edema can be resolved by observing subsequent electrocardiograms of these patients for chronically persisting giant negative T waves 5 and R waves 6 in the mid-precordial leads, and by comparing old and follow-up echocardiograms.…”
Section: Myocardial Apical Hypertrophy and Takotsubo Cardiomyopathymentioning
confidence: 94%