70-year-old blind woman was admitted to hospital because of an initial episode of chest pain lasting 12 h. She had been receiving insulin therapy for diabetes mellitus. An ECG showed ST-segment elevation in the precordial leads (V1-5), I and aVL indicating an anteroseptal acute myocardial infarction (AMI). Emergency coronary arteriography revealed diffuse stenosis of the right and left coronary arteries with severe calcification, and total occlusion at the middle portion of the left anterior descending artery. Blood flow was not apparent in the lesion after balloon inflation because the guide wire had passed through the false lumen. Left ventriculography showed dyskinetic movement at the antero-apex, and hyperkinesis at the other sites (Fig 1A,B). Systolic blood pressure was strictly kept below 120 mmHg by continuous drip infusion of nitroglycerine and oral administration of isosorbide mononitrate (20 mg/day) and imidapril hydrochloride (1.25 mg/day) to prevent rupture of the left ventricle (LV). Echocardiography on the 10th day after onset (Fig 2A,B) showed an aneurysm forming in the antero-apical wall of the LV with moderate pericardial effusion. Along the border between the infarcted and non-infarcted areas, the myocardial wall thickness abruptly decreased and small, freely moving torn pieces were seen (Fig 2A,B). The ratio of the maximal internal diameter of the aneurysm neck (Omax)/the maximal parallel internal diameter of the LV sac (Dmax) was 0.61.The patient underwent an urgent operation on the 13th daybecause of a highly suspected LV pseudoaneurysm. There were 120 ml of slightly bloody effusion in the pericardial cavity and a hemorrhagic infarcted lesion on the surface of the LV apex. Although there was no active bleeding point, fibrin glue and oxycellulose were affixed to this lesion. 1 Two months after the surgery, the patient was readmitted because of dyspnea on mild exertion. The LV volume (left ventricular end-diastolic volume index (LVEDVI): 194 ml/m 2 , left ventricular end-systolic volume index (LVESVI): 134 ml/m 2 , ejection fraction (EF): 31%) had increased to more than twice the volume seen at the onset of AMI (LVEDVI: 89 ml/m 2 , LVESVI: 54 ml/m 2 , EF: 39%) (Fig 1). She successfully underwent Dor's operation to reduce the LV volume. The histological findings of residual small myocyte islands and fibrotic epicardial lesion in the resected A 70-year-old woman with acute myocardial infarction (AMI) had a narrow necked left ventricular (LV) aneurysm and pericardial effusion. Although there had been no obvious sign of pseudoaneurysm at the first operation on the 13th day after onset, LV volume increased so dramatically that dyspnea on mild exertion was induced only 2 months after the onset of AMI. She underwent Dor's operation for the expanded LV aneurysm. The histological findings of the resected tissue, which were fibrotic epicardial lesion with small myocyte islands, indicated a true aneurysm. The ultrasound manifestation of a narrow necked aneurysm with abrupt thinning of the myocardium at t...