Underrated Neurotologic Symptoms

Hoffman, Ronald A.; Brookler, Kenneth H.

doi:10.1002/lary.1978.88.7.1127

Cited by 22 publications

(11 citation statements)

References 9 publications

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“…Typically, such symptoms and slight disorientation develop when walking in 'busy' visual surroundings such as supermarket aisles. The development of these symptoms in some patients with vestibular disorders has long been recognised [28,29]; (see [12] for review) and given various names such as Visuo-Vestibular Mismatch [34], Space and Motion Discomfort [31], or Visual Vertigo [11,23]. This syndrome should not be confused with oscillopsia.…”

Section: Visual Vertigomentioning

confidence: 96%

Vision and vertigo

Bronstein

2004

Journal of Neurology

150

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This review deals with two syndromes, oscillopsia and visual vertigo. Oscillopsia is the illusion of oscillation of the visual surroundings. For diagnosis purposes one should ask, when does the oscillopsia occur? If oscillopsia is only present during head (or whole body) movements, the likely underlying cause is a bilateral defect in the vestibulo-ocular reflex (VOR). The more common causes are post meningitic vestibular damage, gentamicin ototoxicity or bilateral idiopathic vestibular failure. When oscillopsia develops after specific head positions, it is usually due to a positional nystagmus, usually the result of brainstem-cerebellar disease. When the oscillopsia is largely unrelated to head movements, one should ask, is it fairly constant or is it in attacks (paroxysmal)? If the oscillopsia is constant it is usually due to the presence of a clinically observable nystagmus; the most common is downbeat nystagmus but the most visually disabling is pendular nystagmus. If the oscillopsia comes in brief attacks it is usually due to a paroxysmal nystagmus as observed in irritative VIII nerve and brainstem lesions. However, the most common cause of paroxysmal oscillopsia is a non organic condition called voluntary nystagmus. Treatment of oscillopsia is often pharmacological but disappointing; the best chance of success is carbamazepine for paroxysmal disorders secondary to structural vestibular nerve/nuclear lesions.Visual vertigo should not be confused with oscillopsia. It can be defined as dizziness provoked by visual environments with large size (full field) repetitive or moving visual patterns. Patients with visual vertigo report discomfort in supermarkets and when viewing movement of large visual objects, eg crowds, traffic, clouds or foliage. Visual vertigo is present in many patients with a history of a peripheral vestibular disorder, particularly those who are visually dependent (ie subjects who use vision preferentially for postural and space orientation control). Patients with visual vertigo benefit from the addition to their standard vestibular rehabilitation of optic flow (optokinetic) stimuli and exercises involving visuo-vestibular conflict.

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Section: Visual Vertigomentioning

confidence: 96%

Vision and vertigo

Bronstein

2004

Journal of Neurology

150

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“…Typically, such symptoms develop when in busy visual surroundings such as supermarket aisles. The development of these symptoms in some patients with vestibular disorders has long been recognized, [4][5][6] and has been given various names such as visuovestibular mismatch 7,8 or visual vertigo. 9,10 This syndrome should not be confused with oscillopsia.…”

Section: Clinical Picture Of Visual Vertigomentioning

confidence: 99%

Visual Vertigo, Motion Sickness, and Disorientation in Vehicles

2020

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Environmental circumstances that result in ambiguity or conflict with the patterns of sensory stimulation may adversely affect the vestibular system. The effect of this conflict in sensory information may be dizziness, a sense of imbalance, nausea, and motion sickness sometimes even to seemingly minor daily head movement activities. In some, it is not only exposure to motion but also the observation of objects in motion around them such as in supermarket aisles or other places with visual commotion; this can lead to dizziness, nausea, or a feeling of motion sickness that is referred to as visual vertigo. All people with normal vestibular function can be made to experience motion sickness, although individual susceptibility varies widely and is at least partially heritable. Motorists learn to interpret sensory stimuli in the context of the car stabilized by its suspension and guided by steering. A type of motorist's disorientation occurs in some individuals who develop a heightened awareness of perceptions of motion in the automobile that makes them feel as though they may be rolling over on corners and as though they are veering on open highways or in streaming traffic. This article discusses the putative mechanisms, consequences and approach to managing patients with visual vertigo, motion sickness, and motorist's disorientation syndrome in the context of chronic dizziness and motion sensitivity.

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“…1 Although a key symptom of many disorders, AF is generally regarded to be nonspecific with a variety of possible etiologies. [2][3][4][5][6][7][8] Persistent, idiopathic AF has a prevalence of 13.4%, and the second most common classification of patients with AF after eustachian tube dysfunction (ETD). 1 In this study, we retrospectively examined a cohort of patients who presented with isolated, prolonged AF.…”

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confidence: 99%

Migraine‐Related Aural Fullness: A Potential Clinical Entity

Moshtaghi

Ghavami

Mahboubi

et al. 2017

Otolaryngol.--head neck surg.

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In this case series, we set out to describe the clinical entity of isolated, prolonged aural fullness (AF) and its relationship with migraine. Patients with isolated, persistent AF for 6 months or more were included with all possible etiologies ruled out. Migraine dietary and lifestyle changes and medical migraine prophylactic therapy were prescribed to all. Eleven patients were included (mean age, 52 years). Six (54%) patients fulfilled International Headache Society criteria for migraine with or without aura. Changes in perceived sensation of AF using the visual analog scale and quality of life questionnaires resulted in a statically significant improvement (P \ .001, 95% confidence interval [CI], 4.7 to 6.72, and P \.001, 95% CI, 25.3 to 22.7, respectively). As such, an improvement of isolated, prolonged AF with migraine lifestyle changes and prophylactic treatment may suggest an etiological association between migraine and prolonged aural fullness.

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Underrated Neurotologic Symptoms

Cited by 22 publications

References 9 publications

Vision and vertigo

Vision and vertigo

Visual Vertigo, Motion Sickness, and Disorientation in Vehicles

Migraine‐Related Aural Fullness: A Potential Clinical Entity

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