“…Some reports suggest that DMV-mediated vagal activity on cardiac tissue occurs through non-canonical nAChR communication between postganglionic parasympathetic neurons to cardiac tissue. 38 However, since mAChR antagonism abolished DMV light-induced bradycardias, this confirms that DMV neurons induce robust bradycardia through the canonical cardiovagal pathway nAChR→mAChR signaling (and not nAChR→nAChR).…”