Update on the ophthalmic management of facial paralysis

MacIntosh, Peter W.; Fay, Aaron

doi:10.1016/j.survophthal.2018.06.001

Cited by 28 publications

(38 citation statements)

References 52 publications

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“…[1][2][3][4] This correlates with the specific anatomical areas involved in the ipsilateral lower pons, most commonly by stroke. [1][2][3][6][7][8] CN IV palsies typically have excyclotorsion, usually measured with double Maddox rod testing or fundus photography, and a hyperdeviated eye where the hyper-deviation worsens in contralateral gaze and ipsilateral head tilt. 5 In this case report, Parks-Bielschowsky three-step test and double Maddox rod testing confirmed a contralateral (left-sided) CN IV palsy in addition to the other right-sided CN V-VIII palsies, and Horner syndrome clinical findings associated with a Foville syndrome.…”

Section: Discussionmentioning

confidence: 99%

“…5 In this case report, Parks-Bielschowsky three-step test and double Maddox rod testing confirmed a contralateral (left-sided) CN IV palsy in addition to the other right-sided CN V-VIII palsies, and Horner syndrome clinical findings associated with a Foville syndrome. [1][2][3][4][5][6][7][8] Interestingly, cases of Horner syndrome combined with contralateral CN IV palsy have also been reported in the literature. 9,10 As the CN IV nuclei are located at the level of the inferior colliculus in the midbrain, 5 the inferior dorsolateral pontine lesion in this case would not explain the concurrent CN IV palsy.…”

Section: Discussionmentioning

confidence: 99%

“…2019 Dorsolateral pontine syndrome also has been called Foville syndrome and includes CN V, CN VI, CN VII, CN VIII, and oculosympathetic paresis 2013. This correlates with the specific anatomical areas involved in the ipsilateral lower pons, most commonly by stroke 2015…”

Section: Discussionmentioning

confidence: 99%

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Dorsolateral pontine syndrome and contralateral fourth nerve palsy in a post‐stroke patient

Borgman

2020

Clinical and Experimental Optometry

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Section: Discussionmentioning

confidence: 99%

Section: Discussionmentioning

confidence: 99%

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Dorsolateral pontine syndrome and contralateral fourth nerve palsy in a post‐stroke patient

Borgman

2020

Clinical and Experimental Optometry

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“…Se suelen emplear medidas conservadoras como gotas lubricantes para los ojos, también pueden utilizar anteojos protectores para proteger físicamente el ojo de un trauma externo. Los pacientes con exposición y sequedad ocular refractaria deben ser referidos a oftalmología (19).…”

Section: Tratamientounclassified

Parálisis de Bell: Diagnóstico y Tratamiento

Redondo

Lizano

2021

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La parálisis de Bell es un síndrome con afectación del nervio facial, el cual se caracteriza por disfunción muscular de la cara, usualmente unilateral y de etiología idiopática o asociado al virus del Herpes simple de inicio agudo de menos de 72 horas. La parálisis de Bell representa 60-75% de las parálisis faciales unilaterales agudas, sin predilección por lateralidad. Se encuentran varios diagnósticos diferenciales como traumatismos craneoencefálicos, enfermedad de Lyme, virus del Herpes-Zoster, así como neoplasias y eventos cerebrovasculares. Tiene manifestaciones clínicas asociadas a afectación nerviosa muscular como dificultad para sonreír, fruncir el ceño, cerrar los ojos, elevar las cejas. Su diagnóstico es principalmente clínico y el manejo debe ser adecuado y de manera oportuna con corticoesteroides en los primeros 3 días desde su inicio, la terapia antiviral no se utiliza como monoterapia, sin embargo, se puede añadir si hay evidencia de afectación por virus del Herpes, la terapia física complementaria también se ha vuelto un factor clave en la recuperación de la función muscular. Los cuidados del ojo son muy importantes, ya que representan un riesgo de lesión en la córnea por cierre incompleto del párpado. El pronóstico es bueno ya que la mayoría de los pacientes tienen una remisión completa.

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“…Facial nerve palsy may induce facial asymmetry, functional and cosmetic impairment, and therefore can severely affect a patient's quality of life and imposes great psychological and social problems on the individual with the condition [5][6][7][8][9] . The causes of facial nerve palsy are variable and includes idiopathic Bell's palsy, trauma (nonsurgical and surgical), infections (such as herpes zoster, otitis media, leprosy, HIV, tuberculosis, meningitis), stroke and neoplasm [10][11][12][13][14][15] . It is often unilateral but can be bilateral, usually in about 40-75% cases it was unilateral palsy [16] .…”

Section: Introductionmentioning

confidence: 99%

Aetiological Profile of Facial Nerve Palsy Seen in Nigerian Tertiary Hospital

Olajide

Adegbiji

Akinbade

et al. 2020

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Background/Aim: Facial nerve palsy may cause facial asymmetry, functional and cosmetic impairment, and therefore imposes great psychological and social problems on the individual with the condition. The aim of this paper was to highlight the aetiological profile of facial nerve palsy (FNP) in two tertiary institutions in Ekiti, southwest, Nigeria. Methods: This was a retrospective review of patients with facial nerve palsy seen and treated at Ear, Nose & Throat (ENT) clinic. All folders and registers of patients diagnosed with facial nerve palsy from January 2010 to December 2019 in the central, ENT and Dental medical records departments were retrieved and reviewed. The information extracted included the socio-demographic characteristics of the patients, clinical presentation, type and aetiology of FNP, side affected, diagnosis/impression, nature of impairment, type of lesion, onset of the disease, treatment and outcome. Results: Of 76 patients analysed, 48(63.2%) were males and 28(36.8%) were females given a male to female ratio of 1:1.7. Their age ranged between 5 to 72 years with a mean of 39.83 ± 17.58 SD. The age range 21-40 years was most commonly affected, representing 31 (40.8%). The commonest cause of facial nerve paralysis was Bell’s palsy in 32(42.1%), followed by trauma 28(36.9%). Of the 28 (36.9%) that was caused by trauma, road traffic injury constituted 15(53.6%). Half (50.0 %) of the lesion affected right side of the face. Seventy (92.1%) was treated medically. Majority (37.0%) presented within one week of their symptoms. All the patients presented with deviation of mouth, followed by inability to close eye in 70 (92.1%). Higher proportion (88.2%) of our patients had lower motor neuron lesion. Conclusion: This study found that majority of our patients was young adults. Bell’s palsy was a major cause of facial nerve paralysis followed by trauma. Most of our patient presented early and did well on conservative treatment. High index of suspicion is essential especially when patients present with injuries involving head and neck region.

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Update on the ophthalmic management of facial paralysis

Cited by 28 publications

References 52 publications

Dorsolateral pontine syndrome and contralateral fourth nerve palsy in a post‐stroke patient

Dorsolateral pontine syndrome and contralateral fourth nerve palsy in a post‐stroke patient

Parálisis de Bell: Diagnóstico y Tratamiento

Aetiological Profile of Facial Nerve Palsy Seen in Nigerian Tertiary Hospital

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