Vigilance of hemodynamic changes immediately after transferring patients is crucial

Wajima, Zen’ichiro; Shiga, Toshiya; Imanaga, Kazuyuki; Inoue, Tetsuo

doi:10.1007/s00540-013-1568-x

Cited by 4 publications

(4 citation statements)

References 15 publications

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“…The microarray assay was performed as described previously515354. The bioinformatics of Gene Ontology (GO) and Pathway analysis were performed by using the Capital Bio Molecule Annotation System plate analysis as described before55. The gene expression data are available at the Gene Expression Omnibus (GEO) website: http://www.ncbi.nlm.nih.gov/geo/ (accession number GSE59672) (Supplemental Materials and methods).…”

Section: Methodsmentioning

confidence: 99%

Protection against doxorubicin-induced myocardial dysfunction in mice by cardiac-specific expression of carboxyl terminus of hsp70-interacting protein

Wang

Zhang

et al. 2016

Sci Rep

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Carboxyl terminus of Hsp70-interacting protein (CHIP) is a critical ubiquitin ligase/cochaperone to reduce cardiac oxidative stress, inflammation, cardiomyocyte apoptosis and autophage etc. However, it is unclear whether overexpression of CHIP in the heart would exert protective effects against DOX-induced cardiomyopathy. Cardiac-specific CHIP transgenic (CHIP-TG) mice and the wild-type (WT) littermates were treated with DOX or saline. DOX-induced cardiac atrophy, dysfunction, inflammation, oxidative stress and cardiomyocyte apoptosis were significantly attenuated in CHIP-TG mice. CHIP-TG mice also showed higher survival rate than that of WT mice (40% versus 10%) after 10-day administration of DOX. In contrast, knockdown of CHIP by siRNA in vitro further enhanced DOX-induced cardiotoxic effects. Global gene microarray assay revealed that after DOX-treatment, differentially expressed genes between WT and CHIP-TG mice were mainly involved in apoptosis, atrophy, immune/inflammation and oxidative stress. Mechanistically, CHIP directly promotes ubiquitin-mediated degradation of p53 and SHP-1, which results in activation of ERK1/2 and STAT3 pathways thereby ameliorating DOX-induced cardiac toxicity.

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Section: Methodsmentioning

confidence: 99%

Protection against doxorubicin-induced myocardial dysfunction in mice by cardiac-specific expression of carboxyl terminus of hsp70-interacting protein

Wang

Zhang

et al. 2016

Sci Rep

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“…Of the 31 studies analyzed in the review, 17 (55%) (12,18,(22)(23)(24)(25)(26)(27)29,(34)(35)(36)(37)(38)(39)41) referred to the DCO phenomenon, and 14 (45%) (13)(14)(15)(16)(17)(19)(20)(21)28,(30)(31)(32)(33)40) , to DCO. The studies were mostly carried out in countries in South America (n=12;38%) (13)(14)(15)17,19,21,28,(30)(31)(32)(33)40) and in Europe (n=9; 29%) (16,18,22,29,(35)(36)(37)(38)42) , with emphasis on...…”

Section: Resultsmentioning

confidence: 99%

Decreased cardiac output: an integrative review

et al. 2023

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Objective: to identify, in the scientific literature, the defining characteristics and contributing factors (related factors, associated conditions and populations at risk) for nursing diagnosis decreased cardiac output. Method: an integrative literature review, conducted between September and October 2020, with an update in March 2022, in the MEDLINE via PubMed, LILACS, SciELO, CINAHL and EMBASE databases. Using acronym PEO, studies published in the last 10 years in Portuguese, English and Spanish were included. A descriptive analysis was carried out to present the elements mapped in the literature. Results: analysis of 31 articles identified different elements, highlighting 4 new related factors: hyperglycemic stress, prone position, left lateral position, sleep deprivation. Individuals with a history of cardiovascular disease and males were identified as possible populations at risk. Final considerations: the elements for decreased cardiac output, identified in the literature, add evidence that justifies the permanence of this diagnosis in the NANDA-I classification.

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“…O Quadro 2 apresenta a sumarização do processo de caracterização e avaliação do nível de evidência dos estudos selecionados. Dos 31 estudos analisados na revisão, 17 (55%) (12,18,(22)(23)(24)(25)(26)(27)29,(34)(35)(36)(37)(38)(39)41) fizeram referência ao fenômeno DCD, e 14 (45%) (13)(14)(15)(16)(17)(19)(20)(21)28,(30)(31)(32)(33)40) , ao DE DCD. Os estudos foram realizados em sua maioria em países da América do Sul (n=12;38%) (13)(14)(15)17,19,21,28,(30)(31)(32)(33)40) e da Europa (n=9; 29%) (16,18,22,29,(35)(36)(37)…”

Section: Resultsunclassified

Débito cardíaco diminuído: revisão integrativa

et al. 2023

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RESUMO Objetivo: identificar, na literatura científica, as características definidoras e fatores contribuintes (fatores relacionados, condições associadas e populações em risco) para o diagnóstico de enfermagem débito cardíaco diminuído. Método: revisão integrativa da literatura, conduzida entre setembro e outubro de 2020, com atualização em março de 2022, nas bases de dados MEDLINE via PubMed, LILACS, SciELO, CINAHL e EMBASE. Com uso do acrônimo PEO, foram incluídos estudos publicados nos últimos 10 anos em português, inglês e espanhol. Realizouse análise descritiva para apresentar os elementos mapeados na literatura. Resultados: análise de 31 artigos identificou diferentes elementos, com destaque para 4 novos fatores relacionados: estresse hiperglicêmico, posição prona, posição lateral esquerda, privação do sono. Indivíduos com história de doença cardiovascular e do sexo masculino foram apontados como possíveis populações em risco. Considerações finais: os elementos para débito cardíaco diminuído, identificados na literatura, agregam evidências que justificam a permanência desse diagnóstico na classificação da NANDA-I.

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Vigilance of hemodynamic changes immediately after transferring patients is crucial

Cited by 4 publications

References 15 publications

Protection against doxorubicin-induced myocardial dysfunction in mice by cardiac-specific expression of carboxyl terminus of hsp70-interacting protein

Protection against doxorubicin-induced myocardial dysfunction in mice by cardiac-specific expression of carboxyl terminus of hsp70-interacting protein

Decreased cardiac output: an integrative review

Débito cardíaco diminuído: revisão integrativa

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