Virology studies and Bell's palsy

Brodie, S. W.

doi:10.1017/s0022215100087417

Cited by 21 publications

(4 citation statements)

References 8 publications

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“…A viral inflammatory process has been suggested as an etiologic factor in the development of APFP. During the last decade a number of reports have described the association of APFP with viral infection [McCormick, 1972;Adour et al, 1975;Grose et al, 1975;Djupesland et al, 1976;Brodie, 1979;Vahlne et al, 19811. In this study the patient age and sex distribution are in accordance with those of previously reported APFP studies [Adour et al, 19781.…”

Section: Discussionmentioning

confidence: 99%

Specific IgG and IgA antibodies to herpes simplex virus and varicella zoster virus in acute peripheral facial palsy patients

Hadar

Tovi

Sidi

et al. 1983

Journal of Medical Virology

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The role of herpes simplex virus (HSV) and varicella zoster virus (VZV) in acute peripheral facial palsy (APFP) was evaluated in 153 patients. The sera of patients were examined for IgG and IgA antibodies to HSV and VZV by the immunoperoxidase antibody-membrane antigen (IPAMA) technique. The prevalence of IgG and IgA seropositivity to HSV was significantly higher in the APFP patients than in the matched control group. No significant difference was found in the geometric mean titer (GMT) to HSV antibodies in the APFP patients compared to the matched control groups. The prevalence of IgA antibodies to VZV was significantly higher in the APFP patients group than in the matched control group. The GMT for VZV IgG antibodies was significantly higher in the APFP patients than in the matched control group. No significant difference was found in the GMT of VZV IgA antibodies. Eleven of 83 patients for whom paired sera were available had significantly increased or decreased IgG and IgA antibody titers to VZV on subsequent examination. Four of these patients did not show any evidence of zosterian eruption. These studies support the concept that VZV and HSV might have a role in the etiopathogenesis of APFP in some of the patients.

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Section: Discussionmentioning

confidence: 99%

Specific IgG and IgA antibodies to herpes simplex virus and varicella zoster virus in acute peripheral facial palsy patients

Hadar

Tovi

Sidi

et al. 1983

Journal of Medical Virology

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“…Many authors have sought to determine whether Bell's palsy is due to herpes simplex virus [1][2][3][4][5][6][7][8][9][10][11][12][13][14], while more general theories o f infectious origin have prompted others to search for evidence of epidemicity or seasonality [10,[13][14][15][16][17][18][19][20][21][22][23][24][25][26][27][28]. A proposed compromise of the seventh nerve within the fallopian canal through hemody namic or metabolic mechanisms has suggest ed to others that diabetes [18,21,22,26,[29][30][31][32][33][34][35][36][37], hypertension [18,21,22,34,[36][...…”

Section: Introductionmentioning

confidence: 99%

Incidence and Risk Factors for Bell's Palsy in Laredo, Texas: 1974–1982

Brandenburg

Annegers

1993

Neuroepidemiology

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Incidence rates of Bell''s palsy in the predominately Mexican-American population of Laredo, Texas, USA, are higher than those reported elsewhere, reaching 23.5 per 100,000 among males and 32.7 per 100,000 among females. During childbearing years (15–44) the risk of Bell''s palsy was greater for women than for men (incidence density ratio 2.0; 95% confidence interval 1.3–3.1). No evidence for seasonality or epidemicity was obtained. Using prevalence estimates from community surveys in south Texas or the Hispanic Health and Nutrition Survey, standardized prevalence ratios (SPR) calculated for hypertension revealed a 10 or 20% increased risk for women and a 50 or 60% increased risk for men. SPRs calculated for diabetes among men with Bell''s palsy were twice that expected, while risks of 60% or 90% were noted for women. The increased incidence of Bell''s palsy observed in Laredo appears to be due to the high prevalence of diabetes in Mexican-Americans and, to a lesser extent, uncontrolled hypertension.

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“…Later circulatory failure in the vasa nevorum leading to secondary ischaemia (Devrise, 1974), infectious inflammatory processes and autoimmune reactions have all been proposed for the aetiopathogenesis of this condition (Abramski et al, 1975). In different reports serological studies suggest the presence of recent viral inflamation in cases of Bell's palsy (Adour et al, 1975;McCormic, 1975;Brodie, 1979), and in idiopathic bilateral facial palsy (Schuring and Saunders, 1964). The prevalence of complement fixing antibodies and specific immunoglobulin seropositivity to Herpes simplex virus has been found to be significantly higher in cases of facial palsy than in a matched control group (Tovi et al, 1980).…”

Section: Discussionmentioning

confidence: 99%

Recurrent bilateral peripheral facial palsy

Stahl

Tovi

1989

J. Laryngol. Otol.

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Facial nerve paralysis is a common otolaryngological diagnosis. Recurrent unilateral peripheral facial palsy is found in about 7 per cent of the cases. Simultaneous bilateral facial palsy is relatively uncommon and occurs in 0.3–2.0 per cent of cases of facial palsy. Recurrent. simultaneous, bilateral, idiopathic facial palsy to the best of our knowledge has never been reported. A case of recurrent, simultaneous, bilateral, idiopathic facial palsy is presented. No evidence of systemic or local disease was found in both attacks of peripheral facial palsies. The association with states of stress is the only common finding between the two attacks.

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Virology studies and Bell's palsy

Cited by 21 publications

References 8 publications

Specific IgG and IgA antibodies to herpes simplex virus and varicella zoster virus in acute peripheral facial palsy patients

Specific IgG and IgA antibodies to herpes simplex virus and varicella zoster virus in acute peripheral facial palsy patients

Incidence and Risk Factors for Bell's Palsy in Laredo, Texas: 1974–1982

Recurrent bilateral peripheral facial palsy

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