Virus-Cytoskeleton Interaction during Replication of Frog Virus 3

Murti, K.G.; Goorha, Rakesh

doi:10.1007/978-1-4613-1615-2_6

Cited by 5 publications

(2 citation statements)

References 69 publications

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“…FV‐3 inhibits selectively tubulin synthesis, phosphorylates vimentin and modifies actin. The changes are relevant to FV‐3 replication as heat‐inactivated or mutants of FV‐3 do not induce these alterations (Murti and Goorha, 1990). Parenteral inoculation of mice or rats with FV‐3, inactivated FV‐3 or its solubilized proteins induces an acute degenerative hepatitis leading to death in less than 24 h. The pathogenic mechanism seems to be a toxic one because FV‐3 does not multiply at temperatures above 30°C (Kirn et al., 1989).…”

Section: Double‐stranded Dna Virusesmentioning

confidence: 99%

Viruses of Lower Vertebrates

Eßbauer¹,

Ahne

2001

Journal of Veterinary Medicine, Series B

108

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Viruses of lower vertebrates recently became a field of interest to the public due to increasing epizootics and economic losses of poikilothermic animals. These were reported worldwide from both wildlife and collections of aquatic poikilothermic animals. Several RNA and DNA viruses infecting fish, amphibians and reptiles have been studied intensively during the last 20 years. Many of these viruses induce diseases resulting in important economic losses of lower vertebrates, especially in fish aquaculture. In addition, some of the DNA viruses seem to be emerging pathogens involved in the worldwide decline in wildlife. Irido-, herpes- and polyomavirus infections may be involved in the reduction in the numbers of endangered amphibian and reptile species. In this context the knowledge of several important RNA viruses such as orthomyxo-, paramyxo-, rhabdo-, retro-, corona-, calici-, toga-, picorna-, noda-, reo- and birnaviruses, and DNA viruses such as parvo-, irido-, herpes-, adeno-, polyoma- and poxviruses, is described in this review.

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Section: Double‐stranded Dna Virusesmentioning

confidence: 99%

Viruses of Lower Vertebrates

Eßbauer¹,

Ahne

2001

Journal of Veterinary Medicine, Series B

108

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“…The structural integrity of these sites is maintained by intermediate filaments (7-11 nm in diameter) that reorganize from their normal position, radiating from the nucleus, and cluster around virogenic stroma. Fine microfilaments (4-8 nm in diameter) composed of actin break up and reform at the cell surface where they assist in budding and release of virus (Murti et a1., 1985;Murti and Goorha, 1990).…”

Section: Particle Assemblymentioning

confidence: 99%

The Insect Viruses

1998

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viii PREFACE an environmental perspective. Viral diseases in beneficial insects can lead to ecological problems, while diseases of insect pests are often welcomed as a means of reducing economic and medical problems. Furthermore, like the baculoviruses, some of the viruses described in this volume are likely to become valuable tools in the escalating human efforts to control and manipulate the ecosystem.The large DNA viruses are described first, beginning with the entomopoxviruses and iridoviruses, followed by those that rely more heavily on insect vectors for their propagation, and then the small DNA densoviruses. These are followed by the RNA viruses, beginning with an account of the three-dimensional structures of small RNA viruses, followed by chapters describing each of the major families of insect RNA viruses, and ending with a review of the development of arbovirus expression systems and how they may be employed in the future.

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Invertebrate Iridescent Viruses

Williams¹

1998

The Insect Viruses

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Virus-Cytoskeleton Interaction during Replication of Frog Virus 3

Cited by 5 publications

References 69 publications

Viruses of Lower Vertebrates

Viruses of Lower Vertebrates

The Insect Viruses

Invertebrate Iridescent Viruses

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