Volume of the Ventricles in Benign Intracranial Hypertension

Reid, A.; Matheson, M.; Teasdale, Graham M.

doi:10.1016/s0140-6736(80)92889-5

Cited by 51 publications

(21 citation statements)

References 16 publications

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“…Female preponderance is a classic feature of BIH [4. 5, 7] whereas in CSVT [34] there is, as in the present series, no sex preponderance, except for obstetric series [41], The high prevalence of obesity is also a well-known feature of BIH [9,10,18,21,23,25,27,32]. By contrast, These marked differences between the two groups indicate that CSVT should be particularly looked for in males and nonobese females presenting with isolated intracranial hypertension.…”

Section: Discussionsupporting

confidence: 42%

Prospective Study of Cerebral Sinus Venous Thrombosis in Patients Presenting with Benign Intracranial Hypertension

Tehindrazanarivelo¹,

Évrard²,

Schaison³

et al. 1992

Cerebrovasc Dis

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In a prospective study of 24 consecutive patients satisfying all clinical, cerebrospinal fluid and CT scan criteria for benign intracranial hypertension (BIH), angiography showed cerebral venous thrombosis affecting dural sinuses (CSVT) in 6 (25 ± 17%, 95%, confidence limits). When comparing patients with (n = 6) and without CSVT (n = 18), the only statistically significant difference concerned the prevalence of obesity (CSVT = 1/6; BIH = 14/18, p < 0.03). Another trend emerged but did not quite reach statistical significance (sex ratio female/male: CSVT = 3/3; BIH = 17/1, p < 0.07). The present study shows that CSVT can present with all the clinical and radiological criteria of BIH and illustrates the need for angiography or MRI to rule out this possibility, particularly in males and in nonobese females.

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Section: Discussionsupporting

confidence: 42%

Prospective Study of Cerebral Sinus Venous Thrombosis in Patients Presenting with Benign Intracranial Hypertension

Tehindrazanarivelo¹,

Évrard²,

Schaison³

et al. 1992

Cerebrovasc Dis

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“…17 Extraovarian production of estrone may link obesity to IIH. Androstenedione is converted exclusively to estrone by adipocytes 18 and can be stored in fat.…”

Section: Pathophysiologymentioning

confidence: 99%

The relationship between obesity and idiopathic intracranial hypertension

Rowe

1999

Int J Obes

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OBJECTIVES: To evaluate the association between obesity and idiopathic intracranial hypertension (IIH) and establish whether there is a relationship with visual outcome. To calculate the prevalence and degree of obesity in a group of patients with IIH. METHODS: 34 patients (31 female and 3 male) were recruited between 1993 ± 1997 with a diagnosis of IIH. Assessment included visual acuity, visual ®eld assessment (Humphrey automated and Goldmann manual perimetry), fundus assessment and measurement of body mass index (BMI). Obesity was de®ned as a BMI of 30 kgam 2 . RESULTS: 70.5% of patients were obese. The relative risk for obesity and IIH was signi®cant at 8 (95%CI: 2,29). This increased to 17 (95%CI: 5,62) for obese females aged 16 to 24 years and 10 (95%CI: 3,35.5) for obese females aged 25 to 34 years. Morbid obesity (BMI b 40) was signi®cantly associated with poor visual outcome. Serial obesity measures showed generally little change in weight over time and there was no correlation between weight change and visual improvement. CONCLUSIONS: This study has documented a relationship between obesity and IIH. A high degree of obesity was associated with a poor visual function and identi®ed as a risk factor for poor outcome. Obesity may be an aetiological factor in this condition although it is unlikely to be the sole cause but more probably a precipitating factor. Weight loss is recommended although failure to lose weight is common. Any weight reduction programme must therefore be actively encouraged and monitored.

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“…Four important observations need to be explained before accepting the above mechanism as the sole factor responsible for the increased intracranial pressure in pseudotumor cerebri. These are (1) the small or normal size of the lateral ventricles [49][50][51][52] and return of the ventricle to normal size after resolution of symptoms 53 ; (2) the increased cerebral blood volume with normal flow, with an intact arteriolar reflex sensitivity to CO 2 inhalation or hypoxia, suggesting the site of the increased volume to be in the cortical veins 36,[54][55][56] ; (3) evidence of brain edema at histologic examination, 57 hemodynamic studies, 56 and at surgery 37 ; and (4) frequent minute-to-minute fluctuations of CSF pressure and return to the original high level within 1 to 2 hours after therapeutic lumbar puncture. 16,58,59 Why don't the lateral ventricles dilate if the subarachnoid CSF volume is increased in pseudotumor cerebri?…”

Section: Reduced Rate Of Csf Resorptionmentioning

confidence: 99%

Pseudotumor Cerebri (Benign Intracranial Hypertension): Etiopathogenesis, Diagnosis, And Management

Phadke

1988

Ann Saudi Med

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Volume of the Ventricles in Benign Intracranial Hypertension

Cited by 51 publications

References 16 publications

Prospective Study of Cerebral Sinus Venous Thrombosis in Patients Presenting with Benign Intracranial Hypertension

Prospective Study of Cerebral Sinus Venous Thrombosis in Patients Presenting with Benign Intracranial Hypertension

The relationship between obesity and idiopathic intracranial hypertension

Pseudotumor Cerebri (Benign Intracranial Hypertension): Etiopathogenesis, Diagnosis, And Management

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