W. J. Linton

Forman, H. Buxton

doi:10.1093/nq/s6-i.4.79e

Cited by 2 publications

(3 citation statements)

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“…Abell 115 5.2.1. Literature Review A115 has been extensively studied since its discovery as a double X-ray peak by Forman et al (1981). White et al (1997); Shibata et al (1999); Gutierrez & Krawczynski (2005) have also studied its X-ray properties.…”

Section: Resultsmentioning

confidence: 99%

Merging Cluster Collaboration: A Panchromatic Atlas of Radio Relic Mergers

Golovich

Dawson

Wittman

et al. 2019

ApJ

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Golovich et al. (2017b) presents an optical imaging and spectroscopic survey of 29 radio relic merging galaxy clusters. In this paper, we study this survey to identify substructure and quantify the dynamics of the mergers. Using a combined photometric and spectroscopic approach, we identify the minimum number of substructures in each system to describe the galaxy populations and estimate the line of sight velocity difference between likely merging subclusters. We find that the line-of-sight velocity components of the mergers are typically small compared with the maximum three dimensional relative velocity (usually < 1000 km s −1 and often consistent with zero). This suggests that the merger axes of these systems are generally in or near the plane of the sky matching findings in magneto-hydrodynamical simulations. In 28 of the 29 systems we identify substructures in the galaxy population aligned with the radio relic(s) and presumed associated merger induced shock. From this ensemble, we identify eight systems to include in a "gold" sample that is prime for further observation, modeling, and simulation study. Additional papers will present weak lensing mass maps and dynamical modeling for each merging system, ultimately leading to new insight into a wide range of astrophysical phenomena at some of the largest scales in the universe.

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Section: Resultsmentioning

confidence: 99%

Merging Cluster Collaboration: A Panchromatic Atlas of Radio Relic Mergers

Golovich

Dawson

Wittman

et al. 2019

ApJ

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“…The ratios indicated in the figures relate to the number of viable effector cells in the NMS plus C-treated group. The CML assay was performed as described previously [15]. Release of "Cr was measured in triplicate samples, and the percentage of release was computed using the formula cpm supernate x 2 2 X cpm cell lysate + cpm supernate % Release = x 100, the denominator representing total counts/well when the sample size is half volume supernate, and half volume saponine lysate, respectively.…”

Section: Cell-mediated Lympholysis (Cml)mentioning

confidence: 99%

“…Since the Eg chain can only be expressed on the cell membrane in association with the E, chain [17], strain A.TH (like H-2S) does not express the E molecule for lack of associable a chain [17], and BlO.T(6R) (like H-24) produces neither 0 nor associable a chains [18]. The combination A.TL anti-A.TH has an additional disparity at the Qa-Tla region, which also codes for CML determinants, but primary CTL responses are not readily generated against these determinants [15,19,20]. Thus, the only cell surface glycoprotein recognized on cells from these w Ajx stimulator strains is the A molecule, although the formal genetic disparity in combinations 5 and 6 includes the whole I (and S) region.…”

Section: Strain Combinations Used To Generate Ctl Specific For a And mentioning

confidence: 99%

Lyt phenotypes of primary cytotoxic T cells generated across the A and E region of the H‐2 complex

et al. 1981

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Six different cell-mediated lympholysis (CML) combinations were established, four of which generated effector cells against the I-E, and two against the I-A molecule. The cell surface phenotype of effector cells was then determined by depletion of cytotoxic T lymphocyte (CTL) activity with antisera and rabbit complement (C) treatment. Both types of effector cells were completely eliminated by treatment with anti-Thy-1.2 antiserum plus C. Anti-Lyt-1.2 and C depleted anti-A and anti-E killer activity but did not eliminate CTL generated across a whole H-2 difference. One out of three different batches of anti-Lyt-2.2 antiserum did not deplete anti-A killer activity, while it efficiently eliminated CTL generated across the E region or whole H-2 difference. However, two batches of anti-Lyt-2.2 antiserum depleted also anti-A CTL activity. A quantitative difference between anti-A and anti-E CTL in terms of Lyt-2 expression was demonstrated by significant differences in recovery of killer activity, after treatment of these two types of CTL with a wide concentration range of the same anti-Lyt-2.2 antiserum and C. Thus it is concluded that anti-A killer cells have the cell surface phenotype of Thy-1+, Lyt-1, Lyt-2, whereas anti-E CTL are Thy-1+, Lyt-1, Lyt-2. The data are discussed in the context of a possible association of Lyt phenotypes of T cells with the type of MHC antigens they recognize.

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W. J. Linton

Cited by 2 publications

References 0 publications

Merging Cluster Collaboration: A Panchromatic Atlas of Radio Relic Mergers

Merging Cluster Collaboration: A Panchromatic Atlas of Radio Relic Mergers

Lyt phenotypes of primary cytotoxic T cells generated across the A and E region of the H‐2 complex

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