What's in a NAIM?

Chong, Ji Y.; Rowland, Lewis P.

doi:10.1001/archneur.63.2.175

Cited by 35 publications

(2 citation statements)

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“…Although Brain’s original patient did not improve with corticosteroid treatment, most patients who were later described did improve. This led to the renaming of HE by some as “steroid-responsive encephalopathy associated with autoimmune thyroiditis” (SREAT) ( 3 ). The pathogenesis of HE is unknown; however, some evidence of a nonvasculitic process has been reported that supports the term “nonvasculitic autoimmune inflammatory meningoencephalitis” (NAIM) as another denomination of HE ( 4 ).…”

Section: Introductionmentioning

confidence: 99%

Hashimoto's encephalopathy

Ramalho¹,

Castillo²

2011

Radiology Case Reports

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Section: Introductionmentioning

confidence: 99%

Hashimoto's encephalopathy

Ramalho¹,

Castillo²

2011

Radiology Case Reports

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“…This possibility ranges from asymptomatic patients having high antibody titers to a severe encephalopathy patient with a mild rise in antibody titers [7]. Hence, the presence of antithyroid antibodies may be linked to another underlying autoimmune predisposition and has been recently termed as nonvasculitic autoimmune meningoencephalitis (NAIM) [19], which requires further speculation.…”

Section: Discussionmentioning

confidence: 99%

A Case of Euthyroid Steroid-Responsive Encephalopathy With Subacute Dementia

John

Datta

Ovallath

2021

Cureus

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Steroid-responsive encephalopathy associated with autoimmune thyroiditis (SREAT), frequently termed as Hashimoto's encephalopathy (HE), is characterized by reversible encephalopathy with the presence of elevated antithyroid antibodies. The condition was initially described due to its association with Hashimoto's thyroiditis. We report a case of euthyroid HE presenting as subacute dementia. A 50-year-old woman presented with progressive memory decline for six weeks. Thyroid function tests, thyroid ultrasound, and cerebrospinal fluid analysis were unremarkable. Electroencephalogram showed generalized slowing with triphasic waves. On magnetic resonance imaging, T1 weighted images revealed hyperintensity in bilateral basal ganglia. Antithyroglobulin and antithyroid peroxidase were markedly elevated. She improved remarkably on tablet prednisolone 60 mg once daily, confirming the suspicion of steroid-responsive encephalopathy. Thus, we conclude that patients with subacute cognitive decline could be screened for antithyroid antibodies in the dementia workup despite their euthyroid status.

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Prion Diseases and other Rapidly Progressive Dementias

Eggenberger

Murman

2017

Neurodegeneration

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What's in a NAIM?

Cited by 35 publications

References 12 publications

Hashimoto's encephalopathy

Hashimoto's encephalopathy

A Case of Euthyroid Steroid-Responsive Encephalopathy With Subacute Dementia

Prion Diseases and other Rapidly Progressive Dementias

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