Frontal fibrosing alopecia (FFA) is a variant of lichen planopilaris, and it most often occurs in postmenopausal women. 1 It is frequently characterized by progressive scarring alopecia at the frontal and temporal hairline, sideburns, and sometimes eyebrows. 2 The histopathological finding is similar to classic lichen planopilaris, which shows lymphocytic infiltration around the isthmus and infundibulum of the upper part of the hair follicle. 3,4 The true pathogenesis of FFA remains unknown, but there are several proposed mechanisms of this disease, such as innate and adaptive immune responses, epithelial-to-mesenchymal transition, peroxisome proliferationactivated receptorγ dysfunction, and hormonal factors. 5 Some authors suggested the possibility that FFA may be an autoimmune disorder. 6 Moreover, predisposing genetic susceptibility has been observed in FFA patients, especially HLA-B*07:02. 7 The increasing incidence of FFA over the last few decades suggests that environmental factors may relate to the pathogenesis. Recently, questionnaire-based studies from the UK reported association between the use of facial skin care products, particularly sunscreen and moisturizer, and FFA. 8,9 In addition, there was a report of significant hair regrowth along the frontal hairline following discontinuation of sunscreen on the forehead in recalcitrant FFA.The possible hypotheses are that facial care products may enter the follicular infundibulum and trigger an immune reaction, and the use of sunscreen may decrease the immunomodulatory effect of ultraviolet radiation. 10 Most Caucasian populations apply sunscreen to prevent sunburn, skin cancer, and wrinkles, all of which occur less