XVII. Sur l’épilepsie partielle continue (Kojewnikoff)

Radermecker, J; Bogaert, Ludo van

doi:10.1159/000148086

Cited by 7 publications

(2 citation statements)

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“…may be of almost diagnostic distinctiveness (Radermecker, 1949;Radermecker and Macken, 1951 ;Cobb and Hill, 1950;Martin, Macken, and Hess, 1950). But in lipoidosis likewise (Cobb, Martin, and Pampiglione, 1952) a pattern of recurring discharges of varying shape and periodicity may be seen.…”

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confidence: 99%

Rapidly Progressive Cerebral Degeneration (Subacute Vascular Encephalopathy) With Mental Disorder, Focal Disturbances, and Myoclonic Epilepsy

Jones¹,

Nevin²

1954

Journal of Neurology, Neurosurgery & Psychiatry

102

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confidence: 99%

Rapidly Progressive Cerebral Degeneration (Subacute Vascular Encephalopathy) With Mental Disorder, Focal Disturbances, and Myoclonic Epilepsy

Jones¹,

Nevin²

1954

Journal of Neurology, Neurosurgery & Psychiatry

102

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“…Anticonvulsant therapy was started at once, and the seizures ceased after 8 days. The sharply delimited tumor is interesting because many investigators (Kautsky and Stengel 1938, Radermecker and Bogaert 1949, Jensen and Denny-Brown 1966 have emphasized that these patients usually have two lesions at two different levels. In two patients with tumor (Chatrian e t al.…”

Section: Discussionmentioning

confidence: 99%

Epilepsia Partialis Continua with Well‐Delimited Subcortical Frontal Tumor

Botez

Brossard

1974

Epilepsia

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Summary A 28‐yr‐old female presented with epilepsia partialis continua of abrupt onset confined to the left arm. The condition persisted for 8 days after diphenylhydantoin and phenobarbital were begun, leaving slight weakness of the left arm. There was no papilledema. At operation a well‐delimited subcortical astrocytoma of the right precentral and central areas was disclosed. The findings agree with other reports that epilepsia partialis continua usually signifies subcortical lesions that sometimes compress the motor cortex. RESUME H s'agit d'une observation clinique d'une patiente agée de 28 ans qui a présenté soudainement une épilepsie partielle myoclonique continue du membre supérieur gauche. Le syndrome a duré 8 jours. On n'a pas pu mettre en évidence aucun signe d'hypertension intracranienne. AL'opération on a trouvé une astrocytome souscorti‐cal assez bien délimité de L'aire centrale et précentrale droite. Les auteurs concluent que bien que L'origine corticale de L'EPC ne peut être niée, du point de vue strictement pratique, le clinicien doit savoir que dans la plupart des cas L'EPC est déterminée par une ou plusieures lésions souscorticales. Des fois les lésions souscorticales compriment le cortex moteur. ZUSAMMEnfassung Eine 28 Jahre alte Frau zeigte eine p14oUtzlich auftretende Epilepsia partialis continua, die auf den linken Arm beschränkt war. Dieser Zustand dauerte 8 Tage nach Therapiebeginn mit Diphenylhydantoin und Phenobarbital und führte zu einer leichten Schwäche des linken Armes. Es entwickelte sich kein Papillenödem. Bei der Operation wurde ein gut abgegrenztes subcortical gelegenes Astrozytom der rechten präzentralen und zentralen Gegend entdeckt. Die Befunde entsprechen anderen Berichten darüber, dass die Epilepsia partialis continua gewöhnlich auf eine subcortical Läsion hindeutet, die manchmal den motorischen Cortex komprimiert. RESUMEN Una mujer de 28 anos se presentó con epilepsia partialis continua de comienzo brusco, afectando exclusivamente el brazo izquierdo. No tenia edema de papila. En la intervención quirúrgica se extrajo un astrocitoma subcortical de las áreas central y precentral del lado derecho. Este hallazgo está de acuerdo con otras comunicaciones que consideran que la epilepsia partialis continua indica una lesion subcortical que comprime la corteza motora.

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Epilepsia partialis continua associated with nonketotic hyperglycemia: Clinical and biochemical profile of 21 patients

Singh

Strobos

1980

Annals of Neurology

155

100

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In 21 patients, epilepsia partialis continua (EPC) was an early symptom of nonketotic hyperglycemia and occurred during an initial phase of hyponatremia and mild hyperosmolality. EPC persisted for an average of 8 days, and its duration correlated predominantly with the degree of hyponatremia. Depression of consciousness and cessation of seizures occurred with increasing severity of hyperglycemia and hyperosmolality. In 9 patients, EPC was the first symptom leading to the diagnosis of diabetes mellitus. Four patients died of serious associated illness. The majority of the patients had evidence of a localized structural cerebral lesion. Metabolic disturbances including hyperglycemia, mild hyperosmolality, hyponatremia, and lack of ketoacidosis contribute to the development of EPC in areas of focal cerebral damage.

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XVII. Sur l’épilepsie partielle continue (Kojewnikoff)

Cited by 7 publications

References 0 publications

Rapidly Progressive Cerebral Degeneration (Subacute Vascular Encephalopathy) With Mental Disorder, Focal Disturbances, and Myoclonic Epilepsy

Rapidly Progressive Cerebral Degeneration (Subacute Vascular Encephalopathy) With Mental Disorder, Focal Disturbances, and Myoclonic Epilepsy

Epilepsia Partialis Continua with Well‐Delimited Subcortical Frontal Tumor

Epilepsia partialis continua associated with nonketotic hyperglycemia: Clinical and biochemical profile of 21 patients

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