Zur prosopagnosie

Gloning, I; Gloning, K; Hoff, Hans; Tschabitscher, H

doi:10.1016/0028-3932(66)90040-6

Cited by 35 publications

(5 citation statements)

References 21 publications

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“…The intonational aspect of phrases, or rather prosodic expression was impossible to derive from the lower facial region shown in the phrase recognition test. Parenthetically, one can add that difficulties SEX DIFFERENCES IN LIPREADING 107 experienced by individuals with the face recognition disorder (prosopagnosia) have long been attributed to a selective deficit in identification and descr iption of the region around the eyes (Gloning, Gloning, Hoff , & Tschabitscher , 1966) rather than the lower part of the face. Moreover , such explanation is unlikely to account for a superior recognition of visemes by females.…”

Section: Discussionmentioning

confidence: 99%

Sex differences in lipreading

Johnson

Hicks

Goldberg³

et al. 1988

Bull. Psychon. Soc.

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Section: Discussionmentioning

confidence: 99%

Sex differences in lipreading

Johnson

Hicks

Goldberg³

et al. 1988

Bull. Psychon. Soc.

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“…He explained that prosopagnosia is selective in that patients may suffer from the specific inability to recognize familiar faces but remain able to discriminate other complex visual stimulus configurations (including unfamiliar faces). A few cases have been reported in which patients, in addition to their inability to recognize familiar faces, were unable to recognize other visual stimuli such as the faces of cows (Assal et al, 1984;Bomstein et al, 1969), the faces of birds (Bornstein, 1963), and inanimate objects such as cars (Gloning et al, 1966;Macrae and Trolle, 1956).…”

Section: Prosopagnosia and Capgras Delusionmentioning

confidence: 99%

Can we learn from the clinically significant face processing deficits, prosopagnosia and capgras delusion?

Wacholtz¹

1996

Neuropsychol Rev

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This review describes two clinically significant face processing deficits, prosopagnosia and Capgras delusion, and provides new knowledge about the face recognition process by a convergence of empirical findings. These empirical findings are structured around two questions that are reviewed from the perspectives of the two deficits. First is the question of hemispheric specificity, which inquires into the degree of each hemisphere's contribution to the face recognition process. Second is the question of dual neural pathways, which addresses the possibility that the face recognition process proceeds along two parallel pathways in the brain. Findings from the hemispheric specificity studies reinforce the current view that right hemispheric involvement is necessary for face recognition while left hemispheric involvement is minimal. Findings from the dual neural pathways studies reinforce the plausible but yet unproven hypothesis that two neural pathways pass information from the visual association cortex in the occipital lobe toward the temporal lobes and limbic system when faces are seen and recognized. These findings, which also indicate that each of the dual neural pathways carries different, nonredundant information, could be instrumental in showing that the pathways play different roles in the manifestations of the clinically significant face processing deficits, prosopagnosia and Capgras delusion.

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“…limited class of objects certainly is prosopagnosia (Bodamer 1947), the failure to recognize faces (e.g. Pallis 1955;Assal et al 1984;Bornstein and Kidron 1959;Cole and Perez-Cruet 1964;De Renzi 1986a,c;Kay and Levin 1982;Lhermitte and Pillon 1975;Nardelli et al 1982;Shuttleworth et al 1982;Beyn and Knyazeva 1962;Bornstein et al 1969;Cohn et al 1977;Warrington and James 1967;Newcombe 1979;Whiteley and Warrington 1977;Levine 1978;Kertesz 1979;Hécaen andAngelergues 1962, 1963;Benton and van Allen 1972;Benson et al 1974;Benton 1980;Damasio et al 1990b;Gloning et al 1966;Hamsher et al 1979;Landis et al 1986Landis et al , 1988Mazzucchi and Biber 1983;Pevzner et al 1962;Sergent and Villemure 1989;Rondot et al 1967). It seems not to be caused by the fact that faces are more complex and similar to each other than most other visual objects, i.e.…”

Section: Specific Associative Agnosias: Prosopagnosiamentioning

confidence: 99%

Failures of visual analysis: scotoma, agnosia, and neglect

Fahle¹

2003

The Neuropsychology of Vision

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This chapter deals with different types of failure to analyse the visual world, starting with the complete loss of vision, blindness, as well as blindness for circumscribed parts of the visual field, scotomata. It also reports some syndromes characterized by difficulties in perception of and discrimination between different domains or submodalities of vision, while leaving most others intact, such as acquired inability to assess visual motion, or acquired colour blindness caused by cerebral damage (achromatopsia). It then provides an overview of the most astonishing examples of failed visual perception: simultanagnosia; neglect; and Balint's syndrome. These syndromes share the phenomenon that conscious perception of objects in (parts of) the peripheral visual field is absent in spite of intact stimulation of the primary visual cortex by these objects. It also attempts to fill in some gaps of knowledge by means of speculation to produce a consistent taxonomy of symptoms encountered in visual neuropsychology. It combines the theoretical background with clinical findings and with a description of at least some of the methods used to diagnose patients suffering from failures of visual analysis.

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Zur prosopagnosie

Cited by 35 publications

References 21 publications

Sex differences in lipreading

Sex differences in lipreading

Can we learn from the clinically significant face processing deficits, prosopagnosia and capgras delusion?

Failures of visual analysis: scotoma, agnosia, and neglect

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