“…Upon injury, AT2s migrate directionally into the sites of injury and transdifferentiate into AT1s, which are primary regulators of alveolar immunological activities, and AT1s collaborate with AT2s in epithelial repair and regeneration. 5 , 6 , 7 , 8 However, the complicated regulatory mechanism behind this process has not been fully elucidated. Therefore, it is important to further study the mechanisms of pulmonary epithelial repair.…”