Camel adaptation to its arid and semi-arid environment and its genetic make-up created some differences in its enzyme systems compared with other domestic animals. The camel is similar to ruminants in most of the diagnostic enzymes used to evaluate liver functions. However, some differences from other domestic species were noticed in muscular, neuromuscular, pancreatic, bone, drug metabolizing, metalo and antioxidant enzymes in serum and/or tissue. The drug-metabolizing enzymes are low in camel liver and it looks that it has not developed a potent drug metabolizing enzyme system as xenobiotic are low in its natural habitat although its liver was found to be potent in the detoxification of some carcinogens. The low plasma pseudcholinesterase in camel compared with ruminants may explain why it is more vulnerable and less adapted to the toxicity of the organophosphorus compound in agricultural pesticides and herbicides; not previously found in its arid and semiarid habitat. The antioxidant system in the camel looks to be very potent to prevent or reduce oxidative stress which may be another adaptation mechanism to its natural environment. More research is required in serum and tissue enzymes in the camel. There is a great variation in a reference range of diagnostic enzymes in camels. This is due to differences in pre-analytical, analytical and post-analytical procedures and the number of specimens used to define the reference range. Indeed, different laboratories may have the different analytical equipment, different reagents and sources, different specimen's collection and handling procedures, different analytical methods and validation, different storage facilities, different staff qualifications, experience and quality background and the presence or not of a proficiency testing program. Moreover, the physiological, pathological, nutritional, genetical, environmental and geographical factors are of paramount importance in camel enzyme activity. Specifically, the effects of season, sex, age, breed and stage of lactation were emphasized in comparison with other species. Therefore, laboratories should establish their own reference values.

Asymmetry study is realized at alive matter organization different levels beginning from micro-and ending with macro-the most comfortable of which represents population-species which expression is sinistrality. There are so called "left diseases" and other distinguishing features which can be thought as so-called pathological asymmetry. Here are several examples of them: intellectual disability or stuttering (on EEG in the second case) [1, 2], learning developmental disorders [3], dyslexias [4], dysgraphias, attention deficiency and hyperactivity syndrome, autism spectrum disorders [5, 6], mirror writing [7], coordination disorders [8] and apraxias [9], epilepsy (left-handed children get sick on it in earlier age and the disease rate is higher in them comparatively to right-handers or even it is absent in right-handers according to another point of view) [10, 11]. There exists handedness neuropsychiatric pattern [12]. It concerns to schyzophreny, Alzheimer's and Parkinson's diseases dominance in left-handers [13]. Normal brain asymmetry is thought to be connected with VIP gene expression [14]. There is a point of view about pathological right-handedness with temporal epilepsy [15] and heart-vascular problems bigger distribution in dexters. Ambidexters who "can not find which hemisphere to use under