Purpose The aim of this study was to compared the effect of cataract extraction by phacoemulsification and posterior chamber lens implantation on measured intraocular pressure (IOP) using the Goldmann applanation tonometer, the Pascal dynamic contour tonometer (DCT), the Goldmann applanation tonometer (GT) and pneumotonometer (PT) Methods 97 eyes were measured in the operated eye one day before and one day, one week and one month after cataract surgery with the three tonometers, the IOP, ocular pulse amplitude (OPA),and central corneal thickness (CCT) Results Corneal edema induced by phacoemulsification cataract surgery resulted in statistically significant increases in CCT (87,8 μm SD 56,8 ;p< 0,001), Pascal DCT IOP (4,8 mmHg, SD 8,0; p< 0,001),Goldmann IOP (1,4 mmHg, SD 5,1;p<0,015) and OPA (0,9 mmHg, SD 3,5; p< 0,025) but not in pneumotonometry IOP (1,1mmHg, SD 5,3; p=0,065). Changes in IOP measured by GT and PT were less than those measured by the Pascal DCT. The variation between the Pascal DCT (Rho 0,247; p=0,038), and Pneumotonometer (Rho 0,358; p=0,002) was strongly correlated to the change in CCT Conclusion Corneal edema after phacoemulsification cataract surgery increased IOP readings in the three tonometer compared, this increment is bigger in Pascal DCT readings. Changes in CCT are statistically significant associated with increased Pascal DCT IOP readings. Change in corneal biomechanics may in part be responsible for increased in the measured of IOP with Pascal DCT and GT

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74-year-old man with hypertension and diabetes mellitus presented with a three-month history of intermittent orbital pain and progressive vision loss in his left eye, deteriorating to hand motions. The intraocular pressure was in the normal range at 13 mm Hg. Biomicroscopy showed prominent iris neovasculature (Figure 1A). Fluorescein angiography disclosed dot hemorrhages in the midperipheral retina, as well as delayed choroidal and retinal perfusion, indicating fundus ischemia. Intravitreal injection of bevacizumab, an anti-vascular endothelial growth factor agent, achieved rapid regression of iris neovasculature within six days (Figure 1B). For iris neovasculature without retinal vascular diseases, ocular ischemic syndrome should be considered, and total occlusion of the left internal carotid artery was shown on carotid duplex ultrasonography and angiography. Subsequent angioplasty with stenting restored the blood flow (see Appendix 1, available at www.cmaj.ca/lookup/suppl/