This article describes the construction of the Conformity to Feminine Norms Inventory (CFNI), which was designed to assess women's conformity to an array of feminine norms found in the dominant culture in the United States. In addition, we present four studies in which the psychometric properties of the CFNI were examined. In Study 1, factor analysis indicated that the CFNI is comprised of eight distinct factors labeled as Nice in Relationships, Thinness, Modesty, Domestic, Care for Children, Romantic Relationship, Sexual Fidelity, and Invest in Appearance. Results from Study 2 indicated that the CFNI has strong internal consistency estimates and differentiates college women from college men. In addition, Study 2 demonstrated that the CFNI Total score and subscale scores relate to Bem Sex Role Inventory and Feminist Identity Development Scale scores in theoretically consistent patterns. Study 3 indicated that the CFNI Total score and several of the subscales significantly and positively relate to scores on the Eating Disorder Inventory. Finally, Study 4 demonstrated that the CFNI Total score and subscale scores have high test-retest estimates for a 2-3 week period. The discussion focuses on potential uses of the CFNI, limitations to the study, and suggestions for future research.

The authors examined how college men’s conformity to an array of masculinity norms varied as a function of their vocational interests to better understand the gendered context of men’s vocational development. Three hundred ten mostly Caucasian and heterosexual college men completed the Conformity to Masculine Norms Inventory, and their answer to the question “What is your ideal job/career after college?” was classified into one of six high point codes reflecting Holland’s RIASEC interests using the Dictionary of Holland Occupational Titles (1996). Multivariate analysis of variance indicated that college men’s conformity to masculinity norms varied as a function of their vocational interests, and univariate analyses indicated that this was true for 9 of the 11 masculinity norms examined. The discussion addresses the implications of a multidimensional conceptualization of masculinity in understanding men’s vocational development.

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Generalized anxiety disorder is characterized by excessive chronic anxiety in association with many somatic symptoms. The disorder has pervasive effects on quality of life, including work, social and educational aspects and requires long-term therapy. Available studies in patients are the Diagnostic and Statistical Manual of Mental Disorders, third edition-revised and fourth edition, which have defined generalized anxiety disorder and demonstrate the efficacy of benzodiazepines, azapirones, some antidepressants and psychotherapy. Benzodiazepines are effective anxiolytics for short-term use but are accompanied by many adverse events. The antidepressants, paroxetine and venlafaxine (Efexor), have demonstrated efficacy in patients with generalized anxiety disorder with mild side-effect profiles. They have the additional benefit of efficacy in depression, which frequently occurs comorbidly in these patients. Long-term efficacy has been shown with venlafaxine in the treatment of this chronic condition, confirming that as in depression, the goal must not just be remission beyond simple symptom resolution but also on to improved functioning and quality of life. Psychotherapy with applied relaxation, cognitive therapy and cognitive behavioral therapy show the most promise in resolving and maintaining treatment gains in the long-term. These approaches may be useful alone or in combination with adjunctive pharmacotherapy to achieve remission. Based on current evidence, the recommended approach to achieving long-term benefits for patients with generalized anxiety disorder is antidepressant therapy with paroxetine or venlafaxine in combination with cognitive behavioral therapy.

Elevated vasodilatory response (blushing) to social situations is characteristic of social phobia (SP). A relatively unexplored basis for this phenomenon is alteration in underlying vasodilatory mechanisms. To investigate this possibility, we evaluated the vasodilatory response induced by methyl nicotinate (niacin ester derivative) in 31 generalized SP patients and 41 matched healthy volunteers (HV). A patch impregnated with 0, 0.1, 0.5, 1, and 10 mM methyl nicotinate was applied to the forearm or face of subjects for 1 min, followed by 20-min laser Doppler spectroscopy blood flow monitoring. Blood flow stimulation with 1 and 10 mM methyl nicotinate was significantly reduced in SP patients by 35 and 17%, respectively. Induced blood flow was negatively correlated with patients' Leibowitz Social Phobia Scale (LSAS) at 1 and 10 mM doses. Furthermore, the maximal rate of change of vasodilatory reaction was correlated with symptom scores at 1 and 10 mM doses. Induced increases in the arm and face blood flow measurements correlated, supporting the external validity of the former location. Generalized SP patients vasodilate less to topical methyl nicotinate challenges, with effect amplification in severely ill patients. Although the mechanism for this is unclear, we propose desensitization of the prostaglandin-mediated vasodilating system as an explanation.