In athletes, the association between epicardial to endocardial electrophysiological differences and early repolarization ECG pattern can be demonstrated by the vectorial theory of the ventricular activation and repolarization.

50ms (pNN50) e LogAF] apresentaram menor redução durante o 3º estágio no grupo A, e o LogAF, um aumento no último estágio (p=0,027), indicando marcante atividade parassimpática neste grupo. A análise da VFC do grupo A não revelou diferença entre pacientes com maior hipertrofia ou diâmetro atrial. CONCLUSÃO: 1) ocorreu predomínio parassimpático durante estimulação autonômica nos pacientes com CMH; 2) não encontramos correlação entre VFC e as medidas ecocardiográficas analisadas.]]>

No abstract

The results suggested that FDSAECG analysis of the P wave accurately predicted patients whose ADIII therapy will be effective in maintaining the sinus rhythm without frequent recurrence of IPAF.

Objective -The initial site of myocardial infarction (MI) may influence the prevalence of ventricular late potentials (VLP), high-frequency signals, due to the time course of ventricular activation. The prevalence of VLP in a period of more than 2 years after acute MI was assessed focusing on the initially injured wall . Methods - [1][2][3] . Due to the low velocity of conduction, these potentials exceed the duration of ventricular activation and are detected in the ST-segment. The use of statistical techniques, in which 2 to 3 hundred sequential QRS complexes detected on the body surface are independently coherent-averaged, allows amplification and identification of these signals because of the reduction in the intensity of the instrumentation noises 4-7 .The identification of ventricular late potentials in patients with a transmural myocardial infarction is of major interest 3,[8][9][10] . The myocardial remodeling process that follows transmural myocardial infarction is characterized by fibrosis, redistribution of fibers in the injured region, and residual metabolic alterations not only plays a major role in segmentary myocardial function but also provides a favorable environment for the development of reentry circuits 11 .Considering these markers, preliminary studies suggest that transmural myocardial infarctions involving the inferiorinferodorsal wall have a higher prevalence of ventricular late potentials than those involving the anterior-anteroseptal wall. These findings are based on differences related to the start of electrical activation in the affected regions 12,13 . However, studies relating the prevalence of ventricular late potentials after transmural myocardial infarction with the affected wall show some conflicting results. Some studies have focused attention on the occurrence of ventricular late potentials in the first 12 months after transmural myocardial infarction when the myocardial remodeling process is still underway 14 . Data on the prevalence of ventricular late potentials in a late phase (>2 years) of transmural myocardial infarction are very scarce. In this phase, the prevalence of ventricular late potentials reflects not only the natural