The efficiency of boric acid, borax and imidacloprid is evaluated for inhibition of adult house fly emergence (IC values) as dry and liquid baits. In the two cases, imidacloprid has the greatest fatal effect at both IC 50 and IC 90 levels followed by boric acid and then borax. In the liquid formulation, IC 50 and IC 90 values are (0.083 and 2.6%), (0.19 and 2.48%) and (2.6 and 16.9%) for three tested compounds respectively. The relative efficiency for imidacloprid and boric acid compared to borax (the least potent one), imidacloprid and boric acid achieved 31.3 and 13.7 times more suppression of adult emergence than borax. In the solid formulation, IC 50 and IC 90 values are (0.083 -0.67%) followed by boric acid (3.7-11.6%) and borax (5.74-21.1%) respectively. It's clear that imidacloprid 68.8 times and boric acid 1.5 times as toxic as borax. Reasons for differences in manifestation of mortality and possibilities for practical application are discussed. We conclude that the efficiency of tested compounds as liquid baits is higher than it as dry baits.

Background:The association between Hyperuricemia (HUA) and Contrast Induced-Acute Kidney Injury (CI-AKI) has not been extensively studied. The aim of our study was to evaluate the association between HUA and CI-CKD in patients with acute ST-Elevation Myocardial Infarction (STEMI) undergoing Percutanious Coronary Intervention (PCI). Methods:We prospectively studied 146 patients with acute STEMI undergoing PCI. According to serum uric acid (SUA), patients were divided into 2 groups, normouricemic group (Group 1, n=68) and hyperuricemic group (Group 11, n=78). HUA was defined as a SUA concentration of more than 7.0mg/dL in men and 6.0mg/ dL in women. Medical records of both groups of patients were reviewed for the occurrence of AKI. Multi-variate regression analysis was used to define independent risk predictors of AKI.Results: AKI was found in 11 of 78 (14.1%) in Group I1 of patients and in 2 of 68 (2.94%) in Group 1 of patients (p=0.0436), baseline Serum Creatinine (SCr) and Estimated Glomerular Filtration Rate (eGFR) were comparable between the two groups. There was positive significant correlation between admission SUA and SCr 48h after PCI in the studied patients. According to regression analysis, SUA, age, Left Ventricular Ejection Fraction (LVEF), multi-vessel disease and volume of Contrast Media (CM) were found to be independent risk factors of AKI. After adjusting for age, sex, LVEF, multi-vessel disease, and volume of CM, SUA remained as an independent risk factor for CI-AKI. Conclusion:Elevated uric acid was associated with higher risk for AKI in STEMI patients with normal SCr treated with PCI.