Cvek M, Lindvall A-M. External root resorption following bleaching of pulpless teeth with oxygen peroxide. Endod Dent Traumatol 1985; 1: 56-60.Abstract -Eleven teeth in which bleaching with 30% oxygen peroxide was followed by external root resorption are presented. One tooth had no traumatic history, while the others were previously subluxated or luxated in accidental injury. Radiographic appearance of resorptions varied. In 2 teeth the resorptions were only superficial and did not progress during the observation time. In 5 teeth the resorptions were associated with ankylosis. In the remaining 4 teeth the resorption was progressive, bowlshaped and associated with radiolucency in the adjacent alveolar bone. It was suggested that damage to the periodontium, caused by oxygen peroxide at the time of treatment, may heal or be followed by ankylosis and, when complicated by bacterial contamination, develop to a progressive root resorption associated with persistent inflammatory changes in periodontal tissues.
Recurrent parotitis in children is a well-described but rare condition of unknown cause. In this follow-up investigation the long-term course of the disease was studied both clinically and radiographically. In 23 of 25 patients investigated, the clinical symptoms disappeared before the patients were 22 years of age, independent of a given therapy. However, sialographic changes, mostly in the form of sialectasis, were seen on follow-up in the majority of cases, in spite of the fact that the patients were clinically symptom free.
Evaluation of bony changes on the condyles has acceptable reliability and specificity but low sensitivity, whereas the temporal component has low reliability and accuracy. Positive findings often correspond to disease while negative findings do not exclude it. If bony abnormality is suspected in the TMJ, and the panoramic radiograph is negative, tomography may be indicated.
Summary. Three thousand children aged 18 months were screened for dental caries and ongoing breast‐feeding at 46 child welfare centres in different parts of Sweden. Of these, 200 children were selected for a more comprehensive examination, involving investigation of dietary, toothbrushing and sucking habits, use of fluoride, and determination of salivary levels of mutans streptococci and lactobacilli. The children were divided into four groups: group 1: children with caries not being breast‐fed; group 2: children with caries being breast‐fed; group 3: caries‐free children being breast‐fed; group 4: caries‐free gender‐ and age‐matched reference children not being breast‐fed. The results showed that 63 of the children (2.1%) had caries and 61 (2.0%) were still being breast‐fed. Twelve (19.7%) of the 61 children still being breast‐fed had caries compared with 51 (1.7%) of the 2939 children not being breast‐fed; the difference was statistically significant. Children with caries and still being breast‐fed had a mean defs of 5.3, and those with caries not being breast‐fed 4.9; the difference was not statistically significant. Children with caries, irrespective of whether they were being breast‐fed or not, had significantly higher numbers of cariogenic food intakes per day than caries‐free children. Mutans streptococci were detected in 67% of the children and lactobacilli in 13%. Children with detectable mutans streptococci and lactobacilli had significantly more caries than those without. The results indicate that Swedish children with prolonged breast‐feeding have a tendency to establish unsuitable dietary habits which constitutes a risk situation for developing caries at an early age.
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