The protein level in the cerebrospinal fluid (CSF) is an important diagnostic tool and, when abnormal, can provide clinicians with clues to the etiology of a patient's condition. Froin's syndrome has been described in previous literature as the combination of xanthochromia, elevated protein, and hypercoagulated CSF. The pathophysiology behind Froin's syndrome is thought to be due to stagnant CSF causing passive and/or active diffusive processes, resulting in hyperproteinosis and hypercoagulation. We present a case of Froin's syndrome in a patient with cervical spine trauma whose extraordinary level of CSF proteinosis helped raise suspicion for underlying obstructive and infectious etiology.

multicenter feasibility trial of myofascial physical therapy for the treatment of urological chronic pelvic pain syndromes.

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THE SOMATIC CONNECTION The primary outcome measure was pain intensity average during the last 7 days of week 8 and at week 20 (follow-up) as recorded on the VAS. Secondary measures included pain on movement, functional disability, and body awareness. The primary outcome on the pain VAS was significant and clinically relevant at week 8 (P=.001) and at week 20 (P=.003). Significant between-group differences at week 20 were found for pain on movement, functional disability, and body awareness. No serious adverse effects were reported. Although it is difficult to directly compare OCMM with CST because of the differences in training of physical therapists and osteopathic physicians, these data are useful for further research in OCMM.