Introduction: Fall is the most common and serious problem that occurs in Aged ,which has significant effects on their health and quality of life.The purpose of this study was to determine the relationship between fear of falls and depression in Aged diabetic patients in Amol hospitals. Method: This is a descriptive-analytic study that was conducted in 2018. The corpus of study involved 191 Aged diabetic patients aged 60 years and over. Sampling method was available. The instrument for data collection was demographic characteristics, FES-I scales, a fifteen-item questionnaire GDS and AMT and ADL. Data analysis was done by using descriptive and inferential tests and was shown via SPSS software. Results: Most participants (50.3%) were female. The average age of participants was 70.4 ± 6.6 years. Based on the findings, the average of fear of falling was (25 ± 8.7) and average of depression was (6.7 ± 2.1). The results of Spearman's correlation coefficient showed a significant positive correlation between fear of falling and depression (P = 0.001, r = 0.234). So that the fear of falls of the Aged were increased, their depression also increased. Conclusion: The results of this study showed that there is a significant relationship between the fear of falling and depression in Aged people. So, psychological treatments and exercise should be done to reduce their fear of falling.
Severe acute respiratory syndrome coronavirus‐2 (SARS‐CoV‐2) damages multiple organs, including the thyroid, by direct invasion and cell entry via angiotensin‐converting enzyme 2 or indirectly by promoting excessive inflammation in the body. The immune system is a critical factor in antiviral immunity and disease progression. In the context of SARS‐CoV‐2 infection, the immune system may become overly activated, resulting in a shift from regulatory to effector responses, which may subsequently promote the development and progression of autoimmune diseases. The incidence of autoimmune thyroid diseases, such as subacute thyroiditis, Graves' disease, and Hashimoto's thyroiditis, increases in individuals with COVID‐19 infection. This phenomenon may be attributed to aberrant responses of T‐cell subtypes, the presence of autoantibodies, impaired regulatory cell function, and excessive production of inflammatory cytokines, namely interleukin (IL)‐6, IL‐1β, interferon‐γ, and tumor necrosis factor‐α. Therefore, insights into the immune responses involved in the development of autoimmune thyroid disease according to COVID‐19 can help identify potential therapeutic approaches and guide the development of effective interventions to alleviate patients' symptoms.
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