We have observed that NK cell activity when assessed by IFN-γ level measurement was negatively correlating with NLR. This result can be helpful in interpreting or predicting NK cell activity in the clinical environment.

Sodium rechargeable batteries have garnered intensive attention as alternatives to currently used lithium‐ion batteries (LIBs). Among various sodium secondary batteries, a rechargeable Na–SO2 battery deserves to be regarded as one of the most promising sodium battery systems because of its high energy density and safety. Here we report the enhanced rate performance of Na–SO2 battery by introduction of meso/macropores in the carbon electrode using urea as a pore‐forming agent. The resulting meso/macroporous carbon electrode leads to much improved rate capability and structural stability of the electrode in a Na–SO2 battery against mechanical stress/strains induced by the formation of large size insulting discharge product (NaCl). We firmly believe that the urea‐based cost‐effective and scalable process for a meso/macroporous electrode would be one of the most practical ways to improve the performance of Na–SO2 battery to be a candidate for post‐LIB systems.

The present study was undertaken to investigate the anti‑obesity effect of a 50% ethanol extract of Euphorbia supina (ESEE) in high‑fat‑diet (HFD)‑induced obese C57BL/6J mice. Mice were fed a HFD with or without ESEE (2, 10, or 50 mg/kg) or with Garcinia cambogia (positive control) for 6 weeks. ESEE supplementation significantly reduced body, epididymal white adipose tissue (eWAT), and organ weights (P<0.05). ESEE also reduced hepatic steatosis and improved serum lipid profiles. In addition, ESEE significantly reduced serum leptin levels and increased adiponectin levels, and significantly downregulated the mRNA and protein levels of proliferator‑activated receptor γ (PPARγ) and CCAAT/enhancer‑binding protein alpha (C/EPBα) in eWAT and liver tissues (all P<0.05). These results suggested that ESEE supplementation protects against HFD‑induced obesity by downregulating PPARγ and C/EPBα, and that ESEE may be beneficial for the prevention and treatment of obesity and associated diseases.