Anctoderma is a rare disease of unknown etiology, today classified as a subtype of the eircumseribed elastotic disorders. The elinical picture shows atrophic patches loeated mainly on the upper trunk which characteristically give the palpating finger the sensation of a hernial orifiee. Our patient with primary idiopathic anctoderma presented a rarely reported concomitance of the inflammatory type of lesions described by Jadassohn and Peliizzari together with the non-inflammatory Schweninger-Buzzi type. Histologieally and ultrastrueturally, both kinds of lesions revealed similar pictures with a scanty perivascular lymphomononuclear cell infiltrate, with rarefied, partly fragmented elastic fibres and normal eollagen fibrils. These findings indicate that these two types of lesions represent different stages of the same disease and not a simple assoeiation. Furthermore, they support the opinion that the elassification of anetoderma into two separate Jadassohn-Pellizzari and Schweninger-Buzzi types may be unnecessary. After treatment with orai tetraeyciine 1 g/day for 3 weeks, no new lesions appeared; however, no improvement of the already present lesions occurred during a 6-month foilow-up period.