This retrospective case series describes imaging findings in seven dogs and two cats with a presumptive diagnosis of sclerosing encapsulating peritonitis (SEP) between 2014 and 2021. Peritoneal effusion was present in all animal patients. Sonographically, echogenic fluid with or without echogenic intraperitoneal septations, gathered or corrugated bowel loops, and abdominal lymphadenomegaly were suggesting an inflammatory process and the presence of adhesions. Gathering of the bowel with abdominal distension and/or signs of intestinal obstruction were major findings on radiographs. Abdominal fat stranding was an additional finding in animals undergoing a CT examination. Previous surgery, pregnancy, and the presence of a perforating foreign body were potential predisposing causes in 4/9 animals. Peritonitis was septic in 4/9 animals. As SEP is a rare condition but life threatening, this detailed description of imaging findings in a short case series can be useful for a presumptive diagnosis and surgical planning.
An 11-month-old French bulldog was presented for a recurrent fluid-filled mass located ventral to the right pinna. An infected parotid sialocele was diagnosed using ultrasonography, computed tomographic sialography and cytological analysis of the fluid. The dog also had a superficial corneal ulcer because of iatrogenic right facial nerve paralysis secondary to previous attempts by the referring veterinarian to excise the mass surgically. Vegetal foreign bodies (grass awns barbs) were found at the entrance to the right parotid canal and were removed with iris forceps. Bacteriology was positive for Streptococcus canis. Conservative management was initiated after grass awn removal, and the dog was discharged with oral antibiotics (amoxicillin-clavulate orally), ophthalmic antibiotic ointment and artificial tears. Clinical signs related to parotid sialocele resolved, and permanent partial temporal tarsorrhaphy was performed 2 months later. At the 3-year follow-up, the dog had no signs of recurrence of the parotid sialocele, but facial paralysis was still present.
To evaluate clinical features of dogs with cholelithiasis and compare outcomes with medical treatment (MT), surgical treatment (ST) and no treatment (NT). MethodsRetrospective review of medical records of dogs with cholelithiasis identified on abdominal ultrasound (AUS) between 2010-2019. Cases were assigned as clinically affected (CA) or incidentally detected (ID) and divided into MT, ST and NT groups. Biochemical parameters and cholelith location were compared between groups with Mann-Whitney U and Chi-squared testing, respectively. Survival times were compared with Kaplan-Meier survival analysis. Results18 dogs were CA and 20 where ID. CA dogs had significantly higher ALP, GGT and ALT than ID dogs. 8 cases were ST, 15 MT and 15 NT. ST dogs had significantly higher ALP, GGT, ALT and bilirubin than NT dogs. A significantly higher proportion of ST dogs had choleliths in the common bile duct than MT. 17 cases had follow-up AUS: cholelithiasis completely resolved at follow-up in 4/10 MT, 4/5 ST and 1/2 NT; decreased cholelith size or number occurred in 1/10 MT and 1/5 ST. Two MT dogs progressed to require surgery. Median survival time was 457.4 days, with no significant differences in survival between groups. Statement (conclusions)Dogs clinically affected by cholelithiasis have higher ALP, GGT and ALT than dogs with incidental cholelithiasis. 5/10 MT dogs had resolution or improvement of cholelithiasis, showing MT is effective for management of canine cholelithiasis with clinical signs and cholelith location playing a role in treatment decision-making. Survival time was not negatively affected by treatment choice.
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