No abstract

Engineering the structure and chemistry of solid electrolyte interface (SEI) on electrode materials is crucial for rechargeable batteries. Using hard carbon (HC) as a platform material, a correlation between Na + storage performance, and the properties of SEI is comprehensively explored. It is found that a "good" SEI layer on HC may not be directly associated with certain kinds of SEI components, such as NaF and Na 2 O. Whereas, arranging nano SEI components with refined structures constructs the foundation of "good" SEI that enables fast Na + storage and interface stability of HC in Na-ion batteries. A layer-by-layer SEI on HC with inorganic-rich inner layer and tolerant organic-rich outer flexible layer can facilitate excellent rate and cycling life. Besides, SEI layer as the gate for Na + from electrolyte to HC electrode can modulate interfacial crystallographic structures of HC with pillar-solvent that function as "pseudo-SEI" for fast and stable Na + storage in optimal 1 m NaPF 6 -TEGDME electrolytes. Such a layer-by-layer SEI combined with a "pseudo-SEI" layer for HC enables an outstanding rate of 192 mAh g −1 at 2 C and stable cycling over 1100 cycles at 0.5 C. This study provides valuable guidance to improve the electrochemical performance of electrode materials through regulation of SEI in optimal electrolytes.

Mupirocin prophylaxis was effective on preventing ESI and peritonitis due to S. aureus and other organisms in PD patients.

Embryonic lung development requires reciprocal endodermal-mesodermal interactions; mediated by various signaling proteins. Wnt/β-catenin is a signaling protein that exhibits the pivotal role in lung development, injury and repair while aberrant expression of Wnt/β-catenin signaling leads to asthmatic airway remodeling: characterized by hyperplasia and hypertrophy of airway smooth muscle cells, alveolar and vascular damage goblet cells metaplasia, and deposition of extracellular matrix; resulting in decreased lung compliance and increased airway resistance. The substantial evidence suggests that Wnt/β-catenin signaling links embryonic lung development and asthmatic airway remodeling. Here, we summarized the recent advances related to the mechanistic role of Wnt/β-catenin signaling in lung development, consequences of aberrant expression or deletion of Wnt/β-catenin signaling in expansion and progression of asthmatic airway remodeling, and linking early-impaired pulmonary development and airway remodeling later in life. Finally, we emphasized all possible recent potential therapeutic significance and future prospectives, that are adaptable for therapeutic intervention to treat asthmatic airway remodeling.