Fine particulate matter (PM2.5) air pollution exposure is the largest environmental health risk factor in the United States. Here, we link PM2.5exposure to the human activities responsible for PM2.5pollution. We use these results to explore “pollution inequity”: the difference between the environmental health damage caused by a racial–ethnic group and the damage that group experiences. We show that, in the United States, PM2.5exposure is disproportionately caused by consumption of goods and services mainly by the non-Hispanic white majority, but disproportionately inhaled by black and Hispanic minorities. On average, non-Hispanic whites experience a “pollution advantage”: They experience ∼17% less air pollution exposure than is caused by their consumption. Blacks and Hispanics on average bear a “pollution burden” of 56% and 63% excess exposure, respectively, relative to the exposure caused by their consumption. The total disparity is caused as much by how much people consume as by how much pollution they breathe. Differences in the types of goods and services consumed by each group are less important. PM2.5exposures declined ∼50% during 2002–2015 for all three racial–ethnic groups, but pollution inequity has remained high.
Racial-ethnic minorities in the United States are exposed to disproportionately high levels of ambient fine particulate air pollution (PM2.5), the largest environmental cause of human mortality. However, it is unknown which emission sources drive this disparity and whether differences exist by emission sector, geography, or demographics. Quantifying the PM2.5 exposure caused by each emitter type, we show that nearly all major emission categories—consistently across states, urban and rural areas, income levels, and exposure levels—contribute to the systemic PM2.5 exposure disparity experienced by people of color. We identify the most inequitable emission source types by state and city, thereby highlighting potential opportunities for addressing this persistent environmental inequity.
Fine particulate matter (PM2.5) air pollution has been recognized as a major source of mortality in the United States for at least 25 years, yet much remains unknown about which sources are the most harmful, let alone how best to target policies to mitigate them. Such efforts can be improved by employing high-resolution geographically explicit methods for quantifying human health impacts of emissions of PM2.5 and its precursors. Here, we provide a detailed examination of the health and economic impacts of PM2.5 pollution in the United States by linking emission sources with resulting pollution concentrations. We estimate that anthropogenic PM2.5 was responsible for 107,000 premature deaths in 2011, at a cost to society of $886 billion. Of these deaths, 57% were associated with pollution caused by energy consumption [e.g., transportation (28%) and electricity generation (14%)]; another 15% with pollution caused by agricultural activities. A small fraction of emissions, concentrated in or near densely populated areas, plays an outsized role in damaging human health with the most damaging 10% of total emissions accounting for 40% of total damages. We find that 33% of damages occur within 8 km of emission sources, but 25% occur more than 256 km away, emphasizing the importance of tracking both local and long-range impacts. Our paper highlights the importance of a fine-scale approach as marginal damages can vary by over an order of magnitude within a single county. Information presented here can assist mitigation efforts by identifying those sources with the greatest health effects.
Mechanistic air pollution modeling is essential in air quality management, yet the extensive expertise and computational resources required to run most models prevent their use in many situations where their results would be useful. Here, we present InMAP (Intervention Model for Air Pollution), which offers an alternative to comprehensive air quality models for estimating the air pollution health impacts of emission reductions and other potential interventions. InMAP estimates annual-average changes in primary and secondary fine particle (PM2.5) concentrations—the air pollution outcome generally causing the largest monetized health damages–attributable to annual changes in precursor emissions. InMAP leverages pre-processed physical and chemical information from the output of a state-of-the-science chemical transport model and a variable spatial resolution computational grid to perform simulations that are several orders of magnitude less computationally intensive than comprehensive model simulations. In comparisons run here, InMAP recreates comprehensive model predictions of changes in total PM2.5 concentrations with population-weighted mean fractional bias (MFB) of −17% and population-weighted R2 = 0.90. Although InMAP is not specifically designed to reproduce total observed concentrations, it is able to do so within published air quality model performance criteria for total PM2.5. Potential uses of InMAP include studying exposure, health, and environmental justice impacts of potential shifts in emissions for annual-average PM2.5. InMAP can be trained to run for any spatial and temporal domain given the availability of appropriate simulation output from a comprehensive model. The InMAP model source code and input data are freely available online under an open-source license.
Commonly considered strategies for reducing the environmental impact of light-duty transportation include using alternative fuels and improving vehicle fuel economy. We evaluate the air qualityrelated human health impacts of 10 such options, including the use of liquid biofuels, diesel, and compressed natural gas (CNG) in internal combustion engines; the use of electricity from a range of conventional and renewable sources to power electric vehicles (EVs); and the use of hybrid EV technology. Our approach combines spatially, temporally, and chemically detailed life cycle emission inventories; comprehensive, fine-scale state-of-the-science chemical transport modeling; and exposure, concentration-response, and economic health impact modeling for ozone (O 3 ) and fine particulate matter (PM 2.5 ). We find that powering vehicles with corn ethanol or with coal-based or "grid average" electricity increases monetized environmental health impacts by 80% or more relative to using conventional gasoline. Conversely, EVs powered by lowemitting electricity from natural gas, wind, water, or solar power reduce environmental health impacts by 50% or more. Consideration of potential climate change impacts alongside the human health outcomes described here further reinforces the environmental preferability of EVs powered by low-emitting electricity relative to gasoline vehicles.S ociety is in the midst of a great effort to understand and mitigate anthropogenic greenhouse gas (GHG) emissions and their effects on the global climate (1-5). However, GHG damages are not the only environmental impact of human activities, and are often not even the largest. In transportation, for example, non-GHG air pollution damage externalities generally exceed those from climate change (6-8). Here, we explore the air quality impacts of several proposed transportation fuel interventions: liquid biofuels (9), electric vehicles (EVs) powered by conventional and alternative energy sources (3), biomass feedstocks to power EVs (10, 11), compressed natural gas (CNG) powered vehicles (5), and improved vehicle fuel economy.The air quality impacts of biofuels, transportation electrification, CNG vehicles, and improved fuel economy have been studied (refs. 7, 8, and 12-21; results are summarized in Table S1); our work advances prior research by combining estimates of life cycle emissions [i.e., emissions from production ("upstream") and consumption ("tailpipe") of the fuel] with an advanced air quality impact assessment. In addition, we incorporate greater spatial, temporal, and chemical detail than have prior research efforts. We also report non-GHG air quality life cycle impacts of biomass-powered EVs, which to our knowledge have not yet been described.We use a spatially and temporally explicit life cycle inventory model (22) to estimate total fuel supply chain air pollutant emissions for scenarios where 10% of US projected vehicle miles traveled in year 2020 are driven in 1 of 11 types of passenger cars: (i) conventional gasoline powered vehicles (abbrevi...
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