The aim of this in vitro investigation was to compare various roughness and topography measurement methods to characterize the surface quality in several types of resin composites. The initial surface quality of several resin composites was compared. The materials evaluated were of three categories: i) hybrid: TPH Spectrum; ii) reinforced microfill: Micronew and iii) microhybrid: Synergy Duo, Esthet-X, Point.4 and Palfique Estelite. Three Groups of identical disk-shaped specimens (10 x 1.5 mm) were prepared from each material (n = 6) and polished with Soflex discs. Macro-roughness (Ra) was measured with Group 1 by 2-D profilometry. Atomic Force Microscopy (AFM) gave 3-D images and micro-roughness (Ra) of Group 2. Surface optical gloss at 60 degrees was determined for Group 3. Specimens of each material were also studied by scanning electron microscopy. Macro-Ra values (microm) ranged from 0.30 to 0.56. Micro-Ra values ranged from 0.03 to 0.14 and they differed from macro-Ra values in ranking order. Percentage Gloss values ranged from 30.6 to 70.1%. The results revealed that micro-roughness showed a high correlation with gloss values (r = 0.93), whilst macro-roughness did not (r = 0.62). Moreover, the AFM method showed higher capability to distinguish surface roughness compared with the 2-D profilometry and to reveal more detailed definition of surface texture than the examination under SEM.
The reparative mechanisms that operate following carious and traumatic dental injury are critical for pulp survival and involve a series of highly conserved processes. It appears that these processes share genetic programs-linked to cytoskeletal organization, cell movement, and differentiation-that occur throughout embryogenesis. Reactionary dentin is secreted by surviving odontoblasts in response to moderate stimuli, leading to an increase in metabolic activity. In severe injury, necrotic odontoblasts are replaced by other pulp cells, which are able to differentiate into odontoblast-like cells and produce a reparative dentin. This complex process requires the collaborative efforts of cells of different lineage. The behavior of each of the contributing cell types during the phases of proliferation, migration, and matrix synthesis as well as details of how growth factors control wound cell activities are beginning to emerge. In this review, we discuss what is known about the molecular mechanisms involved in dental repair.
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