Research shows a strong link between neuroticism and internalizing psychopathology, such as depression and anxiety. However, it is unclear to what extent meta-mood knowledge (i.e., attention to emotion and emotional clarity) plays a role as a moderator in this relationship. To investigate this, we collected data on meta-mood knowledge, personality traits, depression, and anxiety in a sample of adolescents (N = 244; 53.7% girls) aged 12 to 18 years (M = 14.6, SD = 1.7) from Catalonia, Spain. Regarding the relationship between neuroticism and depression, results showed that emotional clarity buffered this relationship. Although attention to emotion did not, the joint moderating effect of attention to emotion and emotional clarity was significant. The results on the four different combinations of attention to emotion and emotional clarity showed that the relationship between neuroticism and depression was strongest for high attention and low clarity, less strong for low attention and low clarity, and even lower for high attention and high clarity. And importantly, these similar patterns of association disappeared at low attention and high clarity. In contrast to the relationship between neuroticism and depression, we found no statistically significant moderating effects for the relationship between neuroticism and anxiety. Based on these results, we argue the importance of examining individual differences in emotion-based cognition and understanding when the benefits of emotional clarity are associated with fewer disadvantages of excessive attention. These results provide preliminary evidence that the combination of low attention and high clarity may be an adaptive version of emotional self-awareness in relation to neuroticism and depression.

No abstract

As buildings collapse humanity collapses - earthquake in Turkey (February 06, 2023).

Background: Dietary copper restriction in Wilson’s disease is recommended mostly for 1 year or until showing normal liver enzymes. Little is known about the effect of long-term copper restriction on copper and nutritional status in the body. The relationship between daily copper consumption and serum and urine copper parameters, liver enzymes, and dietary contents was investigated. Methods: In this study, 32 pediatric Wilson’s disease patients who had been on treatment at least for 12 months were included. Clinical features, liver enzymes, serum total copper concentrations, non-ceruloplasmin bound copper concentrations, adjusted copper concentrations, 24-hour urine copper excretions, and macro- and micronutrient consumptions were analyzed. Results: In total, 27 patients reported following copper-restricted diets, while daily copper consumption was low only in 7 patients (21.9%). Total copper concentrations and non-ceruloplasmin-bound copper concentrations were low at 78.1% and 53.1%, respectively. All but one adjusted copper concentration were within normal limits. Total copper concentrations, adjusted copper concentration, and non-ceruloplasmin-bound copper concentrations correlated with each other but none correlated with urine copper excretions. Daily copper consumption was inversely correlated with total copper concentrations ( P = .041, r = –0.363) but not correlated with non-ceruloplasmin-bound copper concentrations and adjusted copper concentrations. There was no relationship between liver enzymes and daily copper consumption and serum and urine copper parameters. High fat consumption with low fiber and vitamin B6 was more common in low daily copper consumption group ( P = .033, P = .029, P = .007, respectively). Conclusions: Daily copper consumption may be the least effective or non-effective factor on liver enzymes in Wilson’s disease. Prolonged copper restriction may result in unintentional dietary imbalance. Avoidance of undernutrition and high-fat meals, as well as enrichment of the meals with vitamin B6 and fiber, should be encouraged during copper-restricted diets.

No abstract