Dietary fatty acids are known to modulate fatty acid metabolism in fish. However, the innate capability of fish to bioconvert short chain fatty acids to health promoting long chain fatty acids (LCPUFA) is insufficient to compensate for a reduced dietary intake. While many studies have focused on the dietary regulation of the fatty acid bioconversion pathways, there is little known regarding the effects of the dietary levels of C(18) polyunsaturated fatty acids (PUFA) on fatty acid metabolism. Here, we show a greater degree of apparent enzyme activity (Δ-6 desaturase) in fish fed a diet with higher amounts of dietary C(18) PUFA. In particular, fish receiving high amounts of dietary C(18) PUFA had a greater amount of Δ-6 desaturase activity acting on 18:3n-3 than 18:2n-6. However, with the gradual reduction of dietary C(18) PUFA there was a shift in substrate preference of Δ-6 desaturase from 18:3n-3 to 18:2n-6. This information will provide valuable insight for the implementation of low fish oil diets, which permit the maintenance of n-3 LCPUFA levels in farmed Murray cod.

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The present study examined 12 abnormal psychology textbooks to determine whether Rosenhan’s classic study, “Being sane in insane places,” was covered, and if so, the nature of that coverage. Only 50% covered the study, with all describing the study as demonstrating the biasing power of psychiatric labels. Two key aspects of the study (the diagnoses of schizophrenia and their supposed subsequent influence on the hospital staff’s perception of the pseudopatients’ normal behavior as pathological) were commonly discussed. However, although the study has been heavily criticized, only two texts discussed any criticism of it. Teachers and text authors are urged to become more familiar with the critical literature on this study, and suggestions for class discussions of the study are provided.