A 7‐month‐old, male, entire Weimaraner was referred for surgical repair of a peritoneopericardial diaphragmatic hernia. Thoracic radiographs confirmed a peritoneopericardial diaphragmatic hernia and showed a vesicular emphysema pattern, suggestive of lung lobe torsion. Herniorrhaphy to repair the peritoneopericardial diaphragmatic hernia was performed, and exploratory median sternotomy revealed a torsion of the right middle lung lobe, as well as complete atelectasis of the right caudal lung lobe. Lung lobectomies were performed, and an autologous pericardial flap was used to repair the diaphragmatic defect. A second operation was required because of the suspicion of active bleeding. It was discharged after 6 days of hospitalisation. It is our hypothesis that peritoneopericardial diaphragmatic hernia contributed to the lung lobe torsion due to alterations in the spatial dimensions of the thoracic cavity. Lung lobe torsion has been previously associated with traumatic diaphragmatic herniation in dogs and cats, and has also been reported in a cat with a congenital peritoneopericardial diaphragmatic hernia and chylothorax.

A 1-year-old Bernese Mountain dog presented with an acute onset of left thoracic limb lameness. Magnetic resonance imaging (MRI) of the left shoulder was performed, showing a subchondral bone defect in the caudomedial aspect of the humeral head.In addition, several round hypointense structures were visible in the biceps tendon sheath. A left shoulder arthroscopy was performed, which confirmed an osteochondritic lesion. Exploration of the biceps tendon sheath via a small open approach allowed retrieval of the fragments, which likely migrated from the joint. Histopathology confirmed the structures to be multiple osteochondritic fragments.

An 8-month-old male castrated domestic shorthair was referred with anorexia, polydipsia, lethargy and diarrhoea, which progressed to marked subcutaneous oedema. The cat had hypoalbuminaemia, hypercholesterolaemia, proteinuria and oedema; clinicopathological findings consistent with severe protein-losing nephropathy and nephrotic syndrome. Histopathology of ultrasound-guided percutaneous renal biopsies identified marked podocyte injury with associated focal segmental glomerulosclerosis, a condition rarely reported in cats. The cat fully recovered following medical management with prednisolone and telmisartan, with the resolution of proteinuria.

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