Asthma's sustenance as a global pandemic, across centuries, can be attributed to the lack of an understanding of its workings and the inability of the existing treatment modalities to provide a long lasting cure without major adverse effects. The discovery of statins boosted by a better comprehension of the pathophysiology of asthma in the past few decades has opened up a potentially alternative line of treatment that promises to be a big boon for the asthmatics globally. However, the initial excellent results from the preclinical and animal studies have not borne the results in clinical trials that the scientific world was hoping for. In light of this, this review analyzes the ways by which statins could benefit in asthma via their pleiotropic anti-inflammatory properties and explain some of the queries raised in the previous studies and provide recommendations for future studies in this field.

This article presents a technique to enhance the broadside gain of a CPW fed slot antenna using a single layer metamaterial (MTM) superstrate. A finite array of 3× 3 ring unit cell has been designed on both sides of a dielectric substrate to form the MTM superstrate. The gain enhancement is obtained using the zero‐index property of the metamaterial. The broadside gain enhancement for the proposed antenna is 7.4 dB more in comparison to that of the reference slot antenna. The proposed MTM superstrate loaded antenna provides a minimum overall thickness in the context of using ZIM superstrate for gain enhancement of antennas reported in earlier literatures. The overall thickness of the MTM loaded antenna is 0.13λ0, where λ0 is the free‐space wavelength at the resonance frequency of the antenna. Also, a high efficiency of about 93.2% is obtained in this case. The loading of the MTM superstrate produces a minimal effect on the cross polarization performance of the proposed slot antenna.

Epigenetics deals with the interactions between genes and the immediate cellular environment. These interactions go a long way in shaping up each and every person's individuality. Further, reversibility of epigenetic interactions may offer a dynamic control over the expression of various critical genes. Thus, tweaking the epigenetic machinery may help cause or cure diseases, especially cancer. Therefore, cancer epigenetics, especially at a molecular level, needs to be scrutinised closely, as it could potentially serve as the future pharmaceutical goldmine against neoplastic diseases. However, in view of its rapidly enlarging scope of application, it has become difficult to keep abreast of scientific information coming out of various epigenetic studies directed against cancer. Using this review, we have attempted to shed light on two of the most important mechanisms implicated in cancer, that is, DNA (deoxyribonucleic acid) methylation and histone modifications, and their place in cancer pathogenesis. Further, we have attempted to take stock of the new epigenetic drugs that have emerged onto the market as well as those in the pipeline that offer hope in mankind's fight against cancer.