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Abstract. The brain ischemic mortality rate in Russia occupies the third position. As a result, a recovery period after an ischemic stroke could undermine social and economic well-being of patients and their close relatives. One of the major consequences of a stroke includes the firm-motor defects. Their degree can be reduced with the help of rehabilitation measures intended to revive the motor function of paralyzed limbs and to train a patient to remain firm upright. A stabilographic rehabilitation training apparatus with biofeedback represents one of the variants of the posture training. This training in a playful way helps a patient to improve the balance and firmness indices of the upright position. This rehabilitation method improved considerably the patients' clinical and stabilographic indices of the balance and firmness function in comparison with the patients whose programmes did not include this method. A patient could sense better that he/she was standing on the both lower limbs. The sensitivity in the lower limbs was intensifying or reviving. According to the additional stabilographic control tests, the total scatter of the pressure centre and the scatter in the sagittal plane, the rate of the pressure centre movement were decreasing; Romberg coefficient became normal.

В связи с приближающимся трёхсотлетием со дня рождения Иммануила Канта (1724-1804) в БФУ имени И. Канта в Калининграде в рамках стратегического проекта «Балтийская долина» создаётся уникальная виртуальная реконструкция нескольких улиц Кёнигсберга, по которым ходил немецкий философ. Сложность воспроизведения архитектурного окружения заключается в почти полном отсутствии сохранившихся элементов среды XVIII века и минимуме имеющихся изобразительных источников. Один из маршрутов пролегает от дома И. Канта до книжной лавки И.Я. Кантера (1738-178 6). На этом пути философ мог видеть здание почты на рыночной площади. Согласно проекту 1730 года, в центре фасада предполагалось сделать сложную архитектурно-скульптурную композицию со статуей короля Фридриха Вильгельма I (скульптор И.Х. Майсснер). После сноса здания в 1906-1907 годах декорация была перенесена на опорную стену перед южной стеной замка, дополнена фонтаном и рельефом (скульптор В. Розенберг). От памятника XVIII века в современном Калининграде сохранился небольшой фрагмент в виде полукруглой ниши, завершённой конхой и обрамлённой парными ионическими пилястрами, что позволило снять точные размеры и на основании со­хранившихся чертежей, рисунков и фотографий сделать виртуальную реконструкцию здания в 3D-модели с помощью современных компьютерных методик.

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Massive anoxic brain injury caused by cardiac arrest leads to wakefulness suppression up to coma. The prediction of outcome is based on the analysis of the clinical features and the results of instrumental tests. One of the well-known signs of an unfavorable prognosis is involuntary motor activity, which is most commonly represented by myoclonus. In case of their cortical origin, they are accompanied by epileptiform activity in the electroencephalogram (EEG).Material and methods. We present a case series and literature review concerning a very rare fatal sign, non-rhythmic spontaneous eye opening accompanied by a “burst-suppression” pattern (BS) in the EEG. All patients suffered from transient acute hypotension or arrhythmia that required cardiopulmonary resuscitation (CPR) in three cases. A literature search found only 11 publications describing post-anoxic tonic eye-opening (PATEO).Results. The PATEO with BS was observed for less than a day followed by cessation of brain bioelectric activity in all patients. Only two patients exhibited isolated eye-opening and closing, while the rest had axial and limbs myoclonus just after CPR. In one case, eyelid opening was followed by a clonic movement of the head to the right, the EEG bursts were prolonged and had spike-like morphology. Three patients received antiepileptic and sedative therapy. All patients died in 3-43 days after the fatal cardiovascular event.Visual superposition of bursts in EEG and myogram of m. orbicularis oculi demonstrating identical morphology for EEG and myographic bursts was described for the first time. Our cases and literature review confirm that, regardless of the intensive treatment, patients with PATEO have fatal outcomes.Conclusion: The clinical and electrographic PATEO with BS phenomenon always indicates a lethal prognosis. The origin of PATEO is still under discussion. We suggest that it could be caused by disinhibition of subcortical and stem structures during extensive death of cerebral cortical neurons .